(Q45286126)

11 December 2017

title

Arsenic trioxide (As(2)O(3)) induces apoptosis through activation of Bax in hematopoietic cells (English)

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11 December 2017

6

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11 December 2017

7

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11 December 2017

Yanhua Zheng

1

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11 December 2017

Hirohito Yamaguchi

2

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11 December 2017

Changhai Tian

3

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11 December 2017

Michael W Lee

4

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11 December 2017

Hong Tang

5

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11 December 2017

language of work or name

English

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publication date

1 May 2005

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11 December 2017

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11 December 2017

volume

24

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11 December 2017

issue

20

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11 December 2017

page(s)

3339-3347

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Arsenic induces apoptosis in B-cell leukaemic cell lines in vitro: activation of caspases and down-regulation of Bcl-2 protein

11 December 2017

cites work

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Bax is present as a high molecular weight oligomer/complex in the mitochondrial membrane of apoptotic cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Spontaneous and drug-induced apoptosis is mediated by conformational changes of Bax and Bak in B-cell chronic lymphocytic leukemia

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Reactive oxygen species are required for hyperoxia-induced Bax activation and cell death in alveolar epithelial cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

v-Abl protein tyrosine kinase (PTK) mediated suppression of apoptosis is associated with the up-regulation of Bcl-XL.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Involvement of reactive oxygen species and caspase 3 activation in arsenite-induced apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

VDAC2 inhibits BAK activation and mitochondrial apoptosis

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Modulation of the mitochondrial permeability transition pore by pyridine nucleotides and dithiol oxidation at two separate sites.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Conformational change and mitochondrial translocation of Bax accompany proteasome inhibitor-induced apoptosis of chronic lymphocytic leukemic cells.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Arsenite blocks growth factor induced activation of the MAP kinase cascade, upstream of Ras and downstream of Grb2-Sos

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Mitochondria and apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Granzyme B-mediated cytochrome c release is regulated by the Bcl-2 family members bid and Bax.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Bcl-2 functions in an antioxidant pathway to prevent apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Nonionic detergents induce dimerization among members of the Bcl-2 family

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Bax in murine thymus is a soluble monomeric protein that displays differential detergent-induced conformations

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

BH3-Only proteins-essential initiators of apoptotic cell death

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

A Bax-induced pro-oxidant state is critical for cytochrome c release during programmed neuronal death.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Arsenite induces apoptosis via a direct effect on the mitochondrial permeability transition pore

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Bcl-2 prevents Bax oligomerization in the mitochondrial outer membrane

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Bcl-2 inhibits a Fas-induced conformational change in the Bax N terminus and Bax mitochondrial translocation.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Identification of the protein-protein contact site and interaction mode of human VDAC1 with Bcl-2 family proteins

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Essential role of voltage-dependent anion channel in various forms of apoptosis in mammalian cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Complete remission after treatment of acute promyelocytic leukemia with arsenic trioxide

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

The voltage-dependent anion channel: an essential player in apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Redox aspects of Bcl-2 function.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

The protein kinase PKB/Akt regulates cell survival and apoptosis by inhibiting Bax conformational change

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Bcl-XL protects BimEL-induced Bax conformational change and cytochrome C release independent of interacting with Bax or BimEL.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Arsenic trioxide, a therapeutic agent for APL.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Essential role of the voltage-dependent anion channel (VDAC) in mitochondrial permeability transition pore opening and cytochrome c release induced by arsenic trioxide

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1208484

7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1208484

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Dimensions Publication ID

11 December 2017

1030082170

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1 reference