(Q45300316)

English

Nonlinear decrease over time in N-acetyl aspartate levels in the absence of neuronal loss and increases in glutamine and glucose in transgenic Huntington's disease mice.

scientific article published in May 2000

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scholarly article

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10800956%20AND%20SRC:MED&resulttype=core&format=json

6 November 2019

Nonlinear decrease over time in N-acetyl aspartate levels in the absence of neuronal loss and increases in glutamine and glucose in transgenic Huntington's disease mice (English)

1 reference

Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10800956%20AND%20SRC:MED&resulttype=core&format=json

6 November 2019

Huntington's disease

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Ole A. Andreassen

4

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6 November 2019

2

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6 November 2019

7

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M F Beal

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6 November 2019

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B G Jenkins

1

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6 November 2019

E Kustermann

3

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6 November 2019

R J Ferrante

5

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6 November 2019

B R Rosen

6

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6 November 2019

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publication date

1 May 2000

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6 November 2019

number of pages

12

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Journal of Neurochemistry

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6 November 2019

74

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6 November 2019

5

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6 November 2019

2108-2119

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6 November 2019

Identification of mitochondrial and non-mitochondrial glutaminase within select neurons and glia of rat forebrain by electron microscopic immunocytochemistry

1 reference

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Glutamine and ketone-body metabolism in the gut of streptozotocin-diabetic rats

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Functional evaluation techniques in mitochondrial disorders

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Frontal lobe volume in patients with Huntington's disease

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Cognitive deficits in Huntington's disease are predicted by dopaminergic PET markers and brain volumes

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Transgenic mouse models of neurodegenerative disease caused by CAG/polyglutamine expansions

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Transgenic models of Huntington's disease

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Assignment of resonance in the 1H spectrum of rat brain by two-dimensional shift correlated and J-resolved NMR spectroscopy

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Spatial localization in NMR spectroscopy in vivo

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Age-dependent vulnerability of the striatum to the mitochondrial toxin 3-nitropropionic acid.

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Distribution of phosphate-activated glutaminase, succinic dehydrogenase, pyruvate dehydrogenase and gamma-glutamyl transpeptidase in post-mortem brain from Huntington's disease and agonal cases

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Altered brain neurotransmitter receptors in transgenic mice expressing a portion of an abnormal human huntington disease gene

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Proton MRS in Huntington's disease

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Serial proton magnetic resonance spectroscopy in acute multiple sclerosis lesions

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Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation

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Are neuronal intranuclear inclusions the common neuropathology of triplet-repeat disorders with polyglutamine-repeat expansions?

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Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brain

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Decreased brain N-acetylaspartate in Huntington's disease

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Increased 3-nitrotyrosine and oxidative damage in mice with a human copper/zinc superoxide dismutase mutation.

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Glucose metabolism in brains of diabetic rats.

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Observation of resolved glucose signals in 1H NMR spectra of the human brain at 4 Tesla

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Resolution improvements in in vivo 1H NMR spectra with increased magnetic field strength

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Huntingtin: a single bait hooks many species

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21 January 2018

Mice transgenic for an expanded CAG repeat in the Huntington's disease gene develop diabetes

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21 January 2018

Evidence for impairment of energy metabolism in vivo in Huntington's disease using localized 1H NMR spectroscopy

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Non-invasive neurochemical analysis of focal excitotoxic lesions in models of neurodegenerative illness using spectroscopic imaging

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1H NMR spectroscopy studies of Huntington's disease: correlations with CAG repeat numbers

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Recovery of N-acetylaspartate in corticomotor neurons of patients with ALS after riluzole therapy

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Ataxin-1 nuclear localization and aggregation: role in polyglutamine-induced disease in SCA1 transgenic mice

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N-acetyl-L-aspartate and other amino acid metabolites in Alzheimer's disease brain: a preliminary proton nuclear magnetic resonance study

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Cerebral metabolic disturbances in patients with subacute and chronic diabetes mellitus: detection with proton MR spectroscopy

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Endocrine functions in Huntington's disease. A two-and-a-half years follow-up study

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Cortical and subcortical glucose consumption measured by PET in patients with Huntington's disease.

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Comparison of somatosensory evoked potentials with striatal glucose consumption measured by positron emission tomography in the early diagnosis of Huntington's disease

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Regulation of mammalian brain cell volume

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Aggregation of N-terminal huntingtin is dependent on the length of its glutamine repeats

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Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice

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Instability of highly expanded CAG repeats in mice transgenic for the Huntington's disease mutation.

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Brain pH and lactic acidosis: quantitative analysis of taurine effect

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Ectopically expressed CAG repeats cause intranuclear inclusions and a progressive late onset neurological phenotype in the mouse

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Synthesis of N-acetyl-l-aspartate by rat brain mitochondria and its involvement in mitochondrial/cytosolic carbon transport

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Evidence supporting the existence of an activity-dependent astrocyte-neuron lactate shuttle

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Abnormal Glucose Tolerance and Arginine Tolerance Tests in Huntington’s Disease

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Increased frequency of diabetes mellitus in patients with Huntington's chorea

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Genetic neurodegenerative diseases: the human illness and transgenic models

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Brain neurotransmitter deficits in mice transgenic for the Huntington's disease mutation

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Huntingtin acts in the nucleus to induce apoptosis but death does not correlate with the formation of intranuclear inclusions.

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