(Q46113920)

19 December 2017

title

Heritable disorder resembling neuronal storage disease in mice expressing prion protein with deletion of an alpha-helix (English)

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19 December 2017

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6

Prusiner SB

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19 December 2017

2

DeArmond SJ

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19 December 2017

5

Cohen FE

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19 December 2017

Muramoto T

1

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19 December 2017

Scott M

series ordinal

3

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19 December 2017

Telling GC

series ordinal

4

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19 December 2017

language of work or name

English

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publication date

1 July 1997

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19 December 2017

published in

Nature Medicine

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19 December 2017

volume

3

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19 December 2017

issue

7

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19 December 2017

page(s)

750-755

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19 December 2017

https://scigraph.springernature.com/pub.10.1038/nm0797-750

exact match

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cites work

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Semipreparative chromatographic method to purify the normal cellular isoform of the prion protein in nondenatured form.

21 January 2018

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Conformational transitions in peptides containing two putative alpha-helices of the prion protein.

21 January 2018

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NMR structure of the mouse prion protein domain PrP(121-231)

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Propagation of prions with artificial properties in transgenic mice expressing chimeric PrP genes.

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Recombinant scrapie-like prion protein of 106 amino acids is soluble

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Normal development and behaviour of mice lacking the neuronal cell-surface PrP protein

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An amber mutation of prion protein in Gerstmann-Sträussler syndrome with mutant PrP plaques

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Vascular variant of prion protein cerebral amyloidosis with tau-positive neurofibrillary tangles: the phenotype of the stop codon 145 mutation in PRNP.

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Ecdysone-inducible gene expression in mammalian cells and transgenic mice.

21 January 2018

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High-level expression and characterization of a purified 142-residue polypeptide of the prion protein

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Insoluble wild-type and protease-resistant mutant prion protein in brains of patients with inherited prion disease

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A C-terminal signal prevents secretion of luminal ER proteins

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Transgenic mice expressing hamster prion protein produce species-specific scrapie infectivity and amyloid plaques.

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7 January 2021

Fatal familial insomnia and familial Creutzfeldt-Jakob disease: clinical, pathological and molecular features.

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Epidemiology of Creutzfeldt-Jakob disease in the United States, 1979-1990: analysis of national mortality data

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Proposed three-dimensional structure for the cellular prion protein

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Conversion of alpha-helices into beta-sheets features in the formation of the scrapie prion proteins

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Chimeric prion protein expression in cultured cells and transgenic mice

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7 January 2021

Conversion of truncated and elongated prion proteins into the scrapie isoform in cultured cells

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7 January 2021

Transcriptional activation by tetracyclines in mammalian cells

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7 January 2021

Intracellular accumulation of the cellular prion protein after mutagenesis of its Asn-linked glycosylation sites

1 reference

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7 January 2021

Acquisition of protease resistance by prion proteins in scrapie-infected cells does not require asparagine-linked glycosylation

1 reference

https://api.crossref.org/works/10.1038%2FNM0797-750

7 January 2021

Control of protein exit from the endoplasmic reticulum

1 reference

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7 January 2021

Regional mapping of prion proteins in brain

1 reference

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7 January 2021

Serial transmission in rodents of neurodegeneration from transgenic mice expressing mutant prion protein

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7 January 2021

Identifiers

DOI

10.1038/NM0797-750

1 reference

19 December 2017

1 reference

19 December 2017