(Q46427332)

English

Survivin enhances telomerase activity via up-regulation of specificity protein 1- and c-Myc-mediated human telomerase reverse transcriptase gene transcription

scientific article published in May 2005

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scholarly article

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Europe PubMed Central

22 December 2017

http://europepmc.org/abstract/MED/15817155

Survivin enhances telomerase activity via up-regulation of specificity protein 1- and c-Myc-mediated human telomerase reverse transcriptase gene transcription (English)

1 reference

Europe PubMed Central

22 December 2017

http://europepmc.org/abstract/MED/15817155

author name string

Teruo Endoh

1

1 reference

Europe PubMed Central

22 December 2017

http://europepmc.org/abstract/MED/15817155

Naoki Tsuji

2

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Europe PubMed Central

22 December 2017

http://europepmc.org/abstract/MED/15817155

Koichi Asanuma

3

1 reference

Europe PubMed Central

22 December 2017

http://europepmc.org/abstract/MED/15817155

Atsuhito Yagihashi

4

1 reference

Europe PubMed Central

22 December 2017

http://europepmc.org/abstract/MED/15817155

Naoki Watanabe

5

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Europe PubMed Central

22 December 2017

http://europepmc.org/abstract/MED/15817155

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publication date

1 May 2005

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22 December 2017

http://europepmc.org/abstract/MED/15817155

Experimental Cell Research

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22 December 2017

http://europepmc.org/abstract/MED/15817155

305

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22 December 2017

http://europepmc.org/abstract/MED/15817155

300-311

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22 December 2017

http://europepmc.org/abstract/MED/15817155

2

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Europe PubMed Central

22 December 2017

http://europepmc.org/abstract/MED/15817155

Apoptosis in the pathogenesis and treatment of disease

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Apoptosis: definition, mechanisms, and relevance to disease

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Escaping cell death: survival proteins in cancer

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An anti-apoptotic protein human survivin is a direct inhibitor of caspase-3 and -7

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Survivin as a radioresistance factor in pancreatic cancer.

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A role for survivin in radioresistance of pancreatic cancer cells.

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Expression of survivin in esophageal cancer: correlation with the prognosis and response to chemotherapy

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Survivin as a predictor of cis-diamminedichloroplatinum sensitivity in gastric cancer patients

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Expression of survivin is correlated with cancer cell apoptosis and is involved in the development of human pancreatic duct cell tumors

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Expression of the antiapoptosis gene, survivin, predicts death from recurrent colorectal carcinoma

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A highly conserved repetitive DNA sequence, (TTAGGG)n, present at the telomeres of human chromosomes

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Long G tails at both ends of human chromosomes suggest a C strand degradation mechanism for telomere shortening

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Telomere shortening is proportional to the size of the G-rich telomeric 3'-overhang

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Telomeres, telomerase and cancer: an up-date

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Extension of life-span by introduction of telomerase into normal human cells

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Reconstitution of telomerase activity in normal human cells leads to elongation of telomeres and extended replicative life span

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Recent advances in telomere biology: implications for human cancer.

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Absence of cancer-associated changes in human fibroblasts immortalized with telomerase

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Inhibition of telomerase limits the growth of human cancer cells.

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Telomere dysfunction and telomerase reactivation in human leukemia cell lines after telomerase inhibition by the expression of a dominant-negative hTERT mutant

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Survivin enhances Fas ligand expression via up-regulation of specificity protein 1-mediated gene transcription in colon cancer cells

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Application of photoshop-based image analysis to quantification of hormone receptor expression in breast cancer.

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Complete chromogen separation and analysis in double immunohistochemical stains using Photoshop-based image analysis

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Prognostic value of nuclear survivin expression in oesophageal squamous cell carcinoma

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Sp1 cooperates with c-Myc to activate transcription of the human telomerase reverse transcriptase gene (hTERT).

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Sp1 phosphorylation regulates apoptosis via extracellular FasL-Fas engagement

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Lipopolysaccharide down-regulates Sp1 binding activity by promoting Sp1 protein dephosphorylation and degradation

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Transactivation of gene expression by Myc is inhibited by mutation at the phosphorylation sites Thr-58 and Ser-62.

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GC box binding induces phosphorylation of Sp1 by a DNA-dependent protein kinase

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Modulation of transcription factor Sp1 by cAMP-dependent protein kinase.

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Activation of Sp1-mediated vascular permeability factor/vascular endothelial growth factor transcription requires specific interaction with protein kinase C zeta

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Ras modulates Myc activity to repress thrombospondin-1 expression and increase tumor angiogenesis

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Increased Sp1 phosphorylation as a mechanism of hepatocyte growth factor (HGF/SF)-induced vascular endothelial growth factor (VEGF/VPF) transcription

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Phosphatidylinositol-3-OH kinase as a direct target of Ras

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Ras-GTPase activating protein (GAP): a putative effector for Ras

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Ras-GTPase activating protein inhibition specifically induces apoptosis of tumour cells

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Identification of Aurora kinases as RasGAP Src homology 3 domain-binding proteins

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Aurora B kinase exists in a complex with survivin and INCENP and its kinase activity is stimulated by survivin binding and phosphorylation

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Survivin enhances Aurora-B kinase activity and localizes Aurora-B in human cells

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Fas and perforin pathways as major mechanisms of T cell-mediated cytotoxicity

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The CTL's kiss of death

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Identifiers

10.1016/J.YEXCR.2004.12.014

1 reference

Europe PubMed Central

22 December 2017

http://europepmc.org/abstract/MED/15817155

1 reference

Europe PubMed Central

22 December 2017

http://europepmc.org/abstract/MED/15817155

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