(Q46547417)

23 December 2017

title

Propofol is cardioprotective in a clinically relevant model of normothermic blood cardioplegic arrest and cardiopulmonary bypass (English)

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23 December 2017

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2

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23 December 2017

author name string

Kelvin H H Lim

series ordinal

1

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23 December 2017

Gianni D Angelini

series ordinal

3

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23 December 2017

M-Saadeh Suleiman

series ordinal

4

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23 December 2017

publication date

1 June 2005

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23 December 2017

Experimental Biology and Medicine

published in

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23 December 2017

volume

230

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23 December 2017

issue

6

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23 December 2017

page(s)

413-420

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23 December 2017

Propofol. An update of its use in anaesthesia and conscious sedation

cites work

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Myocardial protection by anesthetic agents against ischemia-reperfusion injury: an update for anesthesiologists

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Propofol attenuates hydrogen peroxide-induced mechanical and metabolic derangements in the isolated rat heart.

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Dose-dependent protection of cardiac function by propofol during ischemia and early reperfusion in rats: effects on 15-F2t-isoprostane formation

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Antioxidant activity of propofol in blood from anaesthetized patients.

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Differential effects of etomidate, propofol, and midazolam on calcium and potassium channel currents in canine myocardial cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Propofol induces a lowering of free cytosolic calcium in myocardial cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Protection by Cyclosporin A of ischemia/reperfusion-induced damage in isolated rat hearts.

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Cyclosporin A binding to mitochondrial cyclophilin inhibits the permeability transition pore and protects hearts from ischaemia/reperfusion injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Mitochondria: a target for myocardial protection.

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Protection of hearts from reperfusion injury by propofol is associated with inhibition of the mitochondrial permeability transition.

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Sevoflurane but not propofol preserves myocardial function in coronary surgery patients

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Propofol attenuates myocardial lipid peroxidation during coronary artery bypass grafting surgery

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Sevoflurane or desflurane anesthesia plus postoperative propofol sedation attenuates myocardial injury after coronary surgery in elderly high-risk patients

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Intermittent antegrade hyperkalaemic warm blood cardioplegia supplemented with magnesium prevents myocardial substrate derangement in patients undergoing coronary artery bypass surgery.

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Plasma propofol concentration and EEG burst suppression ratio during normothermic cardiopulmonary bypass

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Changes in drug plasma concentrations of an extensively bound and highly extracted drug, propofol, in response to altered plasma binding

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Elimination of drugs and toxins during cardiopulmonary bypass

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Purine metabolism and release during cardioprotection with hyperkalemia and hypothermia

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Age-dependent and hypoxia-related differences in myocardial protection during pediatric open heart surgery

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Effects of the anaesthetic propofol on the calcium-induced permeability transition of rat heart mitochondria: direct pore inhibition and shift of the gating potential.

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Mechanisms of inhibition and uncoupling of respiration in isolated rat liver mitochondria by the general anesthetic 2,6-diisopropylphenol.

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Propofol impairment of mitochondrial respiration in isolated perfused guinea pig hearts determined by reflectance spectroscopy.

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Ischaemic preconditioning inhibits opening of mitochondrial permeability transition pores in the reperfused rat heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Inhibition of mitochondrial permeability transition prevents mitochondrial dysfunction, cytochrome c release and apoptosis induced by heart ischemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

The effects of propofol on the contractility of failing and nonfailing human heart muscles.

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

Clinically relevant concentrations of propofol have no effect on adenosine triphosphate-sensitive potassium channels in rat ventricular myocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1177%2F15353702-0323006-09

10.1177/15353702-0323006-09

21 January 2018

Identifiers

DOI

1 reference

23 December 2017

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23 December 2017