(Q44401253)

2 December 2017

title

Ischaemic preconditioning inhibits opening of mitochondrial permeability transition pores in the reperfused rat heart (English)

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2 December 2017

6

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2 December 2017

Sabzali A Javadov

1

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2 December 2017

Samantha Clarke

2

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2 December 2017

Manika Das

3

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2 December 2017

Elinor J Griffiths

4

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2 December 2017

Kelvin H H Lim

5

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2 December 2017

language of work or name

English

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publication date

11 April 2003

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2 December 2017

published in

Journal of Physiology

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2 December 2017

volume

549

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2 December 2017

issue

Pt 2

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2 December 2017

page(s)

513-524

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Matrix volume measurements challenge the existence of diazoxide/glibencamide-sensitive KATP channels in rat mitochondria

2 December 2017

cites work

1 reference

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The effects of ischaemic preconditioning, diazoxide and 5-hydroxydecanoate on rat heart mitochondrial volume and respiration

27 August 2018

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Inhibiting mitochondrial permeability transition pore opening: a new paradigm for myocardial preconditioning?

27 August 2018

1 reference

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K(ATP) channel-independent targets of diazoxide and 5-hydroxydecanoate in the heart

27 August 2018

1 reference

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Sanglifehrin A acts as a potent inhibitor of the mitochondrial permeability transition and reperfusion injury of the heart by binding to cyclophilin-D at a different site from cyclosporin A.

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

The permeability transition pore complex: another view.

27 August 2018

1 reference

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Potassium channel openers protect cardiac mitochondria by attenuating oxidant stress at reoxygenation

27 August 2018

1 reference

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Mitochondrial ATP-sensitive potassium channels attenuate matrix Ca(2+) overload during simulated ischemia and reperfusion: possible mechanism of cardioprotection

27 August 2018

1 reference

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Signal transduction of ischemic preconditioning

27 August 2018

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The KATP channel opener diazoxide protects cardiac myocytes during metabolic inhibition without causing mitochondrial depolarization or flavoprotein oxidation

27 August 2018

1 reference

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Cellular energetics in the preconditioned state: protective role for phosphotransfer reactions captured by 18O-assisted 31P NMR.

27 August 2018

1 reference

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Mitochondrial permeability transition and oxidative stress

27 August 2018

1 reference

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Mitochondria: a target for myocardial protection.

27 August 2018

1 reference

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Reversible ischemic inhibition of F(1)F(0)-ATPase in rat and human myocardium

27 August 2018

1 reference

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Calcium preconditioning inhibits mitochondrial permeability transition and apoptosis

27 August 2018

1 reference

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Bioenergetic consequences of opening the ATP-sensitive K(+) channel of heart mitochondria

27 August 2018

1 reference

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Adenosine A1 receptor activation reduces reactive oxygen species and attenuates stunning in ventricular myocytes

27 August 2018

1 reference

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Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

Dinitrophenol, cyclosporin A, and trimetazidine modulate preconditioning in the isolated rat heart: support for a mitochondrial role in cardioprotection

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

Mitochondrial control of cell death

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

ATP-Sensitive potassium channels: a review of their cardioprotective pharmacology

27 August 2018

1 reference

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Mitochondrial transport of cations: channels, exchangers, and permeability transition.

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

The mitochondrial permeability transition pore and its role in cell death

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

Signal transduction in ischemic preconditioning: the role of kinases and mitochondrial K(ATP) channels

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

Mitochondria: a new target for K channel openers?

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

Cyclosporine A limits myocardial infarct size even when administered after onset of ischemia

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

Elucidating the molecular mechanism of the permeability transition pore and its role in reperfusion injury of the heart

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

Direct effects of diazoxide on mitochondria in pancreatic B-cells and on isolated liver mitochondria

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

Ischemic preconditioning.

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

Cyclosporin A binding to mitochondrial cyclophilin inhibits the permeability transition pore and protects hearts from ischaemia/reperfusion injury

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

Oxidative stress, thiol reagents, and membrane potential modulate the mitochondrial permeability transition by affecting nucleotide binding to the adenine nucleotide translocase

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

Protection by Cyclosporin A of ischemia/reperfusion-induced damage in isolated rat hearts.

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

The regulation of the oxidation of fatty acids and other substrates in rat heart mitochondria by changes in the matrix volume induced by osmotic strength, valinomycin and Ca2+.

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

Inhibition of Ca2(+)-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adeni

27 August 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2342939

Mitochondrial ADP/ATP carrier can be reversibly converted into a large channel by Ca2+

27 August 2018

1 reference

12 December 2020

Reactive oxygen species released from mitochondria during brief hypoxia induce preconditioning in cardiomyocytes

1 reference

12 December 2020

Reversal of permeability transition during recovery of hearts from ischemia and its enhancement by pyruvate

1 reference

12 December 2020

Preconditioning in cardiomyocytes protects by attenuating oxidant stress at reperfusion

1 reference

12 December 2020

Evidence for mitochondrial K ATP channels as effectors of human myocardial preconditioning

1 reference

12 December 2020

Limitations of cyclosporin A inhibition of the permeability transition in CNS mitochondria

1 reference

12 December 2020

Diazoxide, an inhibitor of succinate oxidation

1 reference

12 December 2020

Inhibition of mitochondrial metabolism by the diabetogenic thiadiazine diazoxide. I. Action on succinate dehydrogenase and TCA-cycle oxidations

1 reference

12 December 2020

Inhibition of anoxia-induced injury in heart myocytes by cyclosporin A

1 reference

12 December 2020

Mechanisms of ischemic preconditioning in rat myocardium. Roles of adenosine, cellular energy state, and mitochondrial F1F0-ATPase

1 reference

12 December 2020

10.1113/JPHYSIOL.2003.034231

Identifiers

DOI

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2 December 2017

0 references

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