(Q46856634)

English

Combinatorial action of the HDAC inhibitor trichostatin A and etoposide induces caspase-mediated AIF-dependent apoptotic cell death in non-small cell lung carcinoma cells.

scientific article published on 10 December 2007

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scholarly article

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Europe PubMed Central

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29 December 2019

Combinatorial action of the HDAC inhibitor trichostatin A and etoposide induces caspase-mediated AIF-dependent apoptotic cell death in non-small cell lung carcinoma cells (English)

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:18071312%20AND%20SRC:MED&resulttype=core&format=json

29 December 2019

1 reference

based on heuristic

inferred from title

1 reference

based on heuristic

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apoptotic process

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5

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29 December 2019

author name string

Hajji N

1

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29 December 2019

Wallenborg K

2

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29 December 2019

Vlachos P

3

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29 December 2019

Nyman U

4

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29 December 2019

Joseph B

6

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29 December 2019

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10 December 2007

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29 December 2019

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29 December 2019

27

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29 December 2019

22

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29 December 2019

3134-3144

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29 December 2019

https://scigraph.springernature.com/pub.10.1038/sj.onc.1210976

0 references

Mitochondria as the central control point of apoptosis

1 reference

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Apoptosis-inducing factor determines the chemoresistance of non-small-cell lung carcinomas

1 reference

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Histone deacetylase (HDAC) inhibitor LBH589 increases duration of gamma-H2AX foci and confines HDAC4 to the cytoplasm in irradiated non-small cell lung cancer

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Valproic acid defines a novel class of HDAC inhibitors inducing differentiation of transformed cells

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Bax in murine thymus is a soluble monomeric protein that displays differential detergent-induced conformations

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Defective caspase-3 relocalization in non-small cell lung carcinoma.

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Mitochondrial dysfunction is an essential step for killing of non-small cell lung carcinomas resistant to conventional treatment

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Caspase-8 in apoptosis: the beginning of "the end"?

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Bcl-XL small interfering RNA sensitizes cisplatin-resistant human lung adenocarcinoma cells

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Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics

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Histone deacetylases and cancer: causes and therapies

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Histone deacetylase inhibitors in programmed cell death and cancer therapy

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Breaking the mitochondrial barrier

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Histone deacetylase inhibitors and the promise of epigenetic (and more) treatments for cancer

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Identification and functional significance of genes regulated by structurally different histone deacetylase inhibitors

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Respiratory chain-generated oxidative stress following treatment of leukemic blasts with DNA-damaging agents.

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Bax oligomerization in mitochondrial membranes requires tBid (caspase-8-cleaved Bid) and a mitochondrial protein.

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Suberoylanilide hydroxamic acid combined with gemcitabine enhances apoptosis in non-small cell lung cancer

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Combination phenylbutyrate/gemcitabine therapy effectively inhibits in vitro and in vivo growth of NSCLC by intrinsic apoptotic pathways

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BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death

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Histone deacetylase inhibitors: understanding a new wave of anticancer agents

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Restoring E-cadherin expression increases sensitivity to epidermal growth factor receptor inhibitors in lung cancer cell lines.

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Paclitaxel-induced apoptosis in BJAB cells proceeds via a death receptor-independent, caspases-3/-8-driven mitochondrial amplification loop

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The mitochondrion in apoptosis: how Pandora's box opens

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21 January 2018

Tumor radiosensitivity and apoptosis

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Formation of 50 kbp chromatin fragments in isolated liver nuclei is mediated by protease and endonuclease activation.

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21 January 2018

Apoptosis-inducing factor (AIF): caspase-independent after all.

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Estimation of prognostic value of Bcl-xL gene expression in non-small cell lung cancer

1 reference

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7 January 2021

based on heuristic

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Cleavage of BID during cytotoxic drug and UV radiation-induced apoptosis occurs downstream of the point of Bcl-2 action and is catalysed by caspase-3: a potential feedback loop for amplification of apoptosis-associated mitochondrial cytochrome c rel

1 reference

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7 January 2021

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Modulation of p53, ErbB1, ErbB2, and Raf-1 expression in lung cancer cells by depsipeptide FR901228

1 reference

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based on heuristic

inferred from DOI database lookup

Identifiers

10.1038/SJ.ONC.1210976

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:18071312%20AND%20SRC:MED&resulttype=core&format=json

29 December 2019

1 reference

Europe PubMed Central

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29 December 2019

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