(Q46881121)

26 December 2017

title

ACE inhibition reduces glomerulosclerosis and regenerates glomerular tissue in a model of progressive renal disease (English)

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26 December 2017

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7

Remuzzi G

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26 December 2017

6

Benigni A

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26 December 2017

Remuzzi A

1

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26 December 2017

Gagliardini E

2

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26 December 2017

Sangalli F

3

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26 December 2017

Bonomelli M

4

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26 December 2017

Piccinelli M

5

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26 December 2017

language of work or name

English

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publication date

1 April 2006

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26 December 2017

published in

Kidney International

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26 December 2017

volume

69

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26 December 2017

issue

7

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26 December 2017

page(s)

1124-1130

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26 December 2017

https://scigraph.springernature.com/pub.10.1038/sj.ki.5000060

exact match

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cites work

Prevalence of chronic kidney disease and decreased kidney function in the adult US population: Third National Health and Nutrition Examination Survey

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Control of glomerular hypertension limits glomerular injury in rats with reduced renal mass

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Pathophysiology of Progressive Nephropathies

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

The progression of renal disease

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

The case for intrarenal hypertension in the initiation and progression of diabetic and other glomerulopathies

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Progression, remission, regression of chronic renal diseases

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

ACE inhibition induces regression of proteinuria and halts progression of renal damage in a genetic model of progressive nephropathy.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Effect of angiotensin II antagonism on the regression of kidney disease in the rat.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Enalapril and low protein reverse chronic puromycin aminonucleoside nephropathy

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Regression of sclerosis in aging by an angiotensin inhibition-induced decrease in PAI-1

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Regression of glomerulosclerosis with high-dose angiotensin inhibition is linked to decreased plasminogen activator inhibitor-1

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Reversal of glomerular lesions involves coordinated restructuring of glomerular microvasculature

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Three-dimensional morphometric analysis of segmental glomerulosclerosis in the rat

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Three-dimensional analysis of glomerular morphology in patients with subtotal nephrectomy

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Angiotensin II stimulates extracellular matrix protein synthesis through induction of transforming growth factor-beta expression in rat glomerular mesangial cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Overexpression of chemokines, fibrogenic cytokines, and myofibroblasts in human membranous nephropathy

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Permanent genetic tagging of podocytes: fate of injured podocytes in a mouse model of glomerular sclerosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Platelet-derived growth factor plays a critical role to convert bone marrow cells into glomerular mesangial-like cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Glomerular aging in females is a multi-stage reversible process mediated by phenotypic changes in progenitors.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Angiotensin converting enzyme inhibition ameliorates glomerular filtration of macromolecules and water and lessens glomerular injury in the rat

1 reference

https://api.crossref.org/works/10.1038%2FSJ.KI.5000060

7 January 2021

Identifiers

DOI

10.1038/SJ.KI.5000060

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26 December 2017

1 reference

26 December 2017