(Q46922857)

26 December 2017

title

Matrix metalloproteinase inhibition facilitates cell death in intracerebral hemorrhage in mouse (English)

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26 December 2017

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Mark Grossetete

1

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26 December 2017

Gary A Rosenberg

series ordinal

2

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26 December 2017

publication date

31 October 2007

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26 December 2017

Journal of Cerebral Blood Flow & Metabolism

published in

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26 December 2017

volume

28

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26 December 2017

issue

4

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26 December 2017

page(s)

752-763

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26 December 2017

A metalloproteinase disintegrin that releases tumour-necrosis factor-alpha from cells

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Blood-brain barrier disruption by stromelysin-1 facilitates neutrophil infiltration in neuroinflammation

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The many faces of tumor necrosis factor in stroke

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Tumor necrosis factor-alpha increases in the brain after intracerebral hemorrhage and thrombin stimulation.

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Erythropoietin reduces perihematomal inflammation and cell death with eNOS and STAT3 activations in experimental intracerebral hemorrhage.

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Differential expression of tissue inhibitor of metalloproteinases-3 in cultured astrocytes and neurons regulates the activation of matrix metalloproteinase-2.

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Evidence for apoptosis after intercerebral hemorrhage in rat striatum

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Antisense oligodeoxynucleotide inhibition of tumor necrosis factor-alpha expression is neuroprotective after intracerebral hemorrhage.

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Matrix metalloproteinase expression is related to hemorrhagic transformation after cardioembolic stroke.

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Temporal and spatial profile of apoptotic cell death in transient intracerebral mass lesion of the rat.

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Gelatinase B: a tuner and amplifier of immune functions.

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Intracerebral hemorrhage induces macrophage activation and matrix metalloproteinases

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Increased brain expression of matrix metalloproteinase-9 after ischemic and hemorrhagic human stroke.

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Immunohistochemistry of matrix metalloproteinases in reperfusion injury to rat brain: activation of MMP-9 linked to stromelysin-1 and microglia in cell cultures.

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Effect of synthetic matrix metalloproteinase inhibitors on lipopolysaccharide-induced blood-brain barrier opening in rodents: Differences in response based on strains and solvents

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Metalloproteinase inhibition blocks edema in intracerebral hemorrhage in the rat.

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The design, structure, and clinical update of small molecular weight matrix metalloproteinase inhibitors

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Mmp-9 deficiency enhances collagenase-induced intracerebral hemorrhage and brain injury in mutant mice

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Tissue inhibitor of metalloproteinases-3 and matrix metalloproteinase-3 regulate neuronal sensitivity to doxorubicin-induced apoptosis

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Combination of thrombin and matrix metalloproteinase-9 exacerbates neurotoxicity in cell culture and intracerebral hemorrhage in mice.

21 January 2018

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Matrix metalloproteinase-mediated disruption of tight junction proteins in cerebral vessels is reversed by synthetic matrix metalloproteinase inhibitor in focal ischemia in rat.

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Zymographic measurement of gelatinase activity in brain tissue after detergent extraction and affinity-support purification.

21 January 2018

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The metalloproteinase matrilysin proteolytically generates active soluble Fas ligand and potentiates epithelial cell apoptosis

21 January 2018

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TNF-alpha converting enzyme (TACE) is inhibited by TIMP-3

21 January 2018

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7 January 2021

New alpha-substituted succinate-based hydroxamic acids as TNFalpha convertase inhibitors

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7 January 2021

Intracerebral hemorrhage-induced neuronal death

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https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600572

7 January 2021

Nuclear factor-kappaB and cell death after experimental intracerebral hemorrhage in rats

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600572

7 January 2021

The use of TaqMan RT-PCR assays for semiquantitative analysis of gene expression in CNS tissues and disease models

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https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600572

7 January 2021

Apoptosis as a Form of Cell Death in Intracerebral Hemorrhage

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600572

7 January 2021

Spontaneous intracerebral hemorrhage

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600572

7 January 2021

Identifiers

DOI

10.1038/SJ.JCBFM.9600572

1 reference

26 December 2017

1 reference

26 December 2017