(Q48053769)

8 February 2018

title

Carbon monoxide shifts energetic metabolism from glycolysis to oxidative phosphorylation in endothelial cells (English)

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8 February 2018

oxidative phosphorylation

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Patrycja Kaczara

1

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Kamil Kus

object named as

Kamil Kus

3

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Roberto Motterlini

object named as

Roberto Motterlini

2

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Stefan Chlopicki

object named as

Stefan Chlopicki

6

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author name string

Agnieszka Zakrzewska

4

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8 February 2018

Andrey Y Abramov

5

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8 February 2018

language of work or name

English

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publication date

27 September 2016

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8 February 2018

published in

FEBS Letters

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8 February 2018

volume

590

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8 February 2018

issue

20

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8 February 2018

page(s)

3469-3480

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8 February 2018

Carbon monoxide-releasing molecules: characterization of biochemical and vascular activities.

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Carbon monoxide is a major contributor to the regulation of vascular tone in aortas expressing high levels of haeme oxygenase-1

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Cerebroprotective effects of the CO-releasing molecule CORM-A1 against seizure-induced neonatal vascular injury

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Inhibition of platelet aggregation by carbon monoxide-releasing molecules (CO-RMs): comparison with NO donors

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CO-metal interaction: Vital signaling from a lethal gas

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NPAS2: a gas-responsive transcription factor

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Regulation of ROS production and vascular function by carbon monoxide

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Carbon monoxide rescues mice from lethal sepsis by supporting mitochondrial energetic metabolism and activating mitochondrial biogenesis.

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Carbon monoxide prevents hepatic mitochondrial membrane permeabilization.

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The inhibition of mitochondrial cytochrome oxidase by the gases carbon monoxide, nitric oxide, hydrogen cyanide and hydrogen sulfide: chemical mechanism and physiological significance

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Carbon monoxide signals via inhibition of cytochrome c oxidase and generation of mitochondrial reactive oxygen species

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Myocardial cytochrome oxidase activity is decreased following carbon monoxide exposure

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Effects of carbon monoxide (CO) delivery by a CO donor or hemoglobin on vascular hypoxia inducible factor 1α and mitochondrial respiration

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Carbon monoxide expedites metabolic exhaustion to inhibit tumor growth

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[Mn(CO)4{S2CNMe(CH2CO2H)}], a new water-soluble CO-releasing molecule.

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Carbon monoxide released by CORM-401 uncouples mitochondrial respiration and inhibits glycolysis in endothelial cells: A role for mitoBKCa channels.

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Measurement of mitochondrial NADH and FAD autofluorescence in live cells.

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Visualization of ATP levels inside single living cells with fluorescence resonance energy transfer-based genetically encoded indicators

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The relationship between mitochondrial state, ATP hydrolysis, [Mg2+]i and [Ca2+]i studied in isolated rat cardiomyocytes.

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Cell metabolism affects selective vulnerability in PINK1-associated Parkinson's disease

21 January 2018

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Dopamine induced neurodegeneration in a PINK1 model of Parkinson's disease

21 January 2018

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Detection of 2-hydroxyethidium in cellular systems: a unique marker product of superoxide and hydroethidine.

21 January 2018

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A positive/negative ion-switching, targeted mass spectrometry-based metabolomics platform for bodily fluids, cells, and fresh and fixed tissue

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Principles of targeting endothelial cell metabolism to treat angiogenesis and endothelial cell dysfunction in disease

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Carbon monoxide modulates apoptosis by reinforcing oxidative metabolism in astrocytes: role of Bcl-2.

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Reductive glutamine metabolism is a function of the α-ketoglutarate to citrate ratio in cells.

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Regulation of substrate utilization by the mitochondrial pyruvate carrier

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Reduced methylation of PFKFB3 in cancer cells shunts glucose towards the pentose phosphate pathway

21 January 2018

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Role of PFKFB3-driven glycolysis in vessel sprouting

21 January 2018

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Endothelial PFKFB3 plays a critical role in angiogenesis

21 January 2018

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Mitochondria in health and disease: perspectives on a new mitochondrial biology

21 January 2018

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Mitochondrial membrane potential regulates matrix configuration and cytochrome c release during apoptosis

21 January 2018

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Different responses of astrocytes and neurons to nitric oxide: the role of glycolytically generated ATP in astrocyte protection.

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21 January 2018

Identifiers

DOI

10.1002/1873-3468.12434

1 reference

8 February 2018

1 reference

8 February 2018