(Q48224518)

9 February 2018

title

Voltage-dependent anion channel involved in the α-synuclein-induced dopaminergic neuron toxicity in rats. (English)

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9 February 2018

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Lingling Lu

1

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9 February 2018

Chunyan Zhang

2

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9 February 2018

Qing Cai

3

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9 February 2018

Qiang Lu

4

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9 February 2018

Chunli Duan

5

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9 February 2018

Yuangang Zhu

6

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9 February 2018

Hui Yang

7

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9 February 2018

publication date

4 January 2013

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9 February 2018

Acta Biochimica et Biophysica Sinica

published in

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9 February 2018

volume

45

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9 February 2018

issue

3

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9 February 2018

page(s)

170-178

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9 February 2018

Mutation in the alpha-synuclein gene identified in families with Parkinson's disease

cites work

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Immunoelectron-microscopic demonstration of NACP/alpha-synuclein-epitopes on the filamentous component of Lewy bodies in Parkinson's disease and in dementia with Lewy bodies

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

alpha-Synuclein in filamentous inclusions of Lewy bodies from Parkinson's disease and dementia with lewy bodies

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Alpha-synuclein overexpression reduces gap junctional intercellular communication in dopaminergic neuroblastoma cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Direct membrane association drives mitochondrial fission by the Parkinson disease-associated protein alpha-synuclein

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Localization of alpha-synuclein to mitochondria within midbrain of mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Involvment of cytosolic and mitochondrial GSK-3beta in mitochondrial dysfunction and neuronal cell death of MPTP/MPP-treated neurons

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Cell death pathways in Parkinson's disease: role of mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Endoplasmic reticulum and mitochondria interplay mediates apoptotic cell death: relevance to Parkinson's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Mitochondrial pathology in Parkinson's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

New insights into the role of mitochondrial dysfunction and protein aggregation in Parkinson's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Mitophagy: the latest problem for Parkinson's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Glutamate interacts with VDAC and modulates opening of the mitochondrial permeability transition pore

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Complexes between porin, hexokinase, mitochondrial creatine kinase and adenylate translocator display properties of the permeability transition pore. Implication for regulation of permeability transition by the kinases

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Role of mitochondrial permeability transition pores in mitochondrial autophagy.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Extensive nuclear localization of alpha-synuclein in normal rat brain neurons revealed by a novel monoclonal antibody

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Overexpression of alpha-synuclein in rat substantia nigra results in loss of dopaminergic neurons, phosphorylation of alpha-synuclein and activation of caspase-9: resemblance to pathogenetic changes in Parkinson's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

alpha-synuclein promotes mitochondrial deficit and oxidative stress

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Mitochondrial import and accumulation of alpha-synuclein impair complex I in human dopaminergic neuronal cultures and Parkinson disease brain

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

What is the mitochondrial permeability transition pore?

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Role of the mitochondrial membrane permeability transition in cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Mitochondrial α-synuclein accumulation impairs complex I function in dopaminergic neurons and results in increased mitophagy in vivo

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

The role of autophagy in Parkinson's disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

SNCA (α-synuclein)-induced toxicity in yeast cells is dependent on sirtuin 2 (Sir2)-mediated mitophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

Role of mitochondrial inner membrane permeabilization in necrotic cell death, apoptosis, and autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

The Ca2+-induced membrane transition in mitochondria. I. The protective mechanisms

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

The Ca2+-induced membrane transition in mitochondria

21 January 2018

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https://api.crossref.org/works/10.1093%2FABBS%2FGMS114

21 January 2018

Identifiers

DOI

10.1093/ABBS/GMS114

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9 February 2018

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9 February 2018