(Q48259447)

English

Selective impairment of fast anterograde axonal transport in the peripheral nerves of asymptomatic transgenic mice with a G93A mutant SOD1 gene.

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Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/10082867

Selective impairment of fast anterograde axonal transport in the peripheral nerves of asymptomatic transgenic mice with a G93A mutant SOD1 gene. (English)

1 reference

Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/10082867

1

1 reference

Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/10082867

author name string

Y Itoyama

2

1 reference

Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/10082867

K Abe

3

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Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/10082867

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1 February 1999

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Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/10082867

1 reference

Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/10082867

819

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Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/10082867

120-131

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Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/10082867

1-2

1 reference

Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/10082867

Ischemic delayed neuronal death. A mitochondrial hypothesis.

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Protective effect of a novel free radical scavenger, OPC-14117, on wobbler mouse motor neuron disease

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Induction of nitrotyrosine-like immunoreactivity in the lower motor neuron of amyotrophic lateral sclerosis

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Familial amyotrophic lateral sclerosis (ALS) in Japan associated with H46R mutation in Cu/Zn superoxide dismutase gene: a possible new subtype of familial ALS

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ALS, SOD and peroxynitrite

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Superoxide dismutase concentration and activity in familial amyotrophic lateral sclerosis

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Morphometric and biochemical studies of peripheral nerves in amyotrophic lateral sclerosis

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Interfering with the runners

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A novel brain ATPase with properties expected for the fast axonal transport motor

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Molecular motors in the nervous system

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Fast axonal transport alterations in amyotrophic lateral sclerosis (ALS) and in parathyroid hormone (PTH)-treated axons

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Fast axonal transport in amyotrophic lateral sclerosis: an intra-axonal organelle traffic analysis

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Defective axonal transport in a transgenic mouse model of amyotrophic lateral sclerosis

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Progressive neuronopathy in transgenic mice expressing the human neurofilament heavy gene: a mouse model of amyotrophic lateral sclerosis

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7 January 2021

Neuropathological changes in two lines of mice carrying a transgene for mutant human Cu,Zn SOD, and in mice overexpressing wild type human SOD: a model of familial amyotrophic lateral sclerosis (FALS).

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A low expressor line of transgenic mice carrying a mutant human Cu,Zn superoxide dismutase (SOD1) gene develops pathological changes that most closely resemble those in human amyotrophic lateral sclerosis

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Neurofibrillary axonal swellings and amyotrophic lateral sclerosis

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Amyotrophic lateral sclerosis and structural defects in Cu,Zn superoxide dismutase

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Pathogenesis of two axonopathies does not require axonal neurofilaments.

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Increased 3-nitrotyrosine and oxidative damage in mice with a human copper/zinc superoxide dismutase mutation.

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Variants of the heavy neurofilament subunit are associated with the development of amyotrophic lateral sclerosis

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Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

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The molecular mechanism of organelle transport along microtubules: the identification and characterization of KIFs (kinesin superfamily proteins).

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Kinesin and dynein superfamily proteins and the mechanism of organelle transport

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Kinesin associates with anterogradely transported membranous organelles in vivo

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Brain dynein (MAP1C) localizes on both anterogradely and retrogradely transported membranous organelles in vivo.

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Neuromuscular function impairment is not caused by motor neurone loss in FALS mice: an electromyographic study

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7 January 2021

A mutant neurofilament subunit causes massive, selective motor neuron death: implications for the pathogenesis of human motor neuron disease

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Identification of a microtubule-based cytoplasmic motor in the nematode C. elegans.

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Neurofilament subunit NF-H modulates axonal diameter by selectively slowing neurofilament transport

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Retrograde transport by the microtubule-associated protein MAP 1C.

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Motor neurons in Cu/Zn superoxide dismutase-deficient mice develop normally but exhibit enhanced cell death after axonal injury

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7 January 2021

Transgenic mice expressing an altered murine superoxide dismutase gene provide an animal model of amyotrophic lateral sclerosis

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Analysis of the KSP repeat of the neurofilament heavy subunit in familiar amyotrophic lateral sclerosis

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Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

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SOD1 mutation is associated with accumulation of neurofilaments in amyotrophic lateral sclerosis.

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Impairment of fast axonal transport in the proximal axons of anterior horn neurons in amyotrophic lateral sclerosis

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Transgenic mice carrying a human mutant superoxide dismutase transgene develop neuronal cytoskeletal pathology resembling human amyotrophic lateral sclerosis lesions

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Identification of a novel force-generating protein, kinesin, involved in microtubule-based motility

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Mechanisms of Fast and Slow Axonal Transport

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Sequence variants in human neurofilament proteins: absence of linkage to familial amyotrophic lateral sclerosis

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An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria

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Increased expression of neurofilament subunit NF-L produces morphological alterations that resemble the pathology of human motor neuron disease

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Neurofilaments and orthograde transport are reduced in ventral root axons of transgenic mice that express human SOD1 with a G93A mutation

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7 January 2021

Identifiers

10.1016/S0006-8993(98)01351-1

1 reference

Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/10082867

1 reference

Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/10082867

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