(Q48287915)

10 February 2018

review article

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title

Transgenic mice expressing Alzheimer amyloid precursor proteins. (English)

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10 February 2018

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Hsiao K

1

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10 February 2018

publication date

1 November 1998

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10 February 2018

published in

Experimental Gerontology

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10 February 2018

volume

33

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10 February 2018

page(s)

883-889

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10 February 2018

issue

7-8

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10 February 2018

Lack of apolipoprotein E dramatically reduces amyloid beta-peptide deposition.

cites work

1 reference

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7 January 2021

Accelerated amyloid deposition in the brains of transgenic mice coexpressing mutant presenilin 1 and amyloid precursor proteins.

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7 January 2021

Release of excess amyloid beta protein from a mutant amyloid beta protein precursor.

1 reference

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7 January 2021

Genetic modification of the phenotypes produced by amyloid precursor protein overexpression in transgenic mice.

1 reference

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7 January 2021

Increased amyloid-beta42(43) in brains of mice expressing mutant presenilin 1

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7 January 2021

Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

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7 January 2021

Early Alzheimer disease-like histopathology increases in frequency with age in mice transgenic for beta-APP751.

1 reference

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7 January 2021

Accelerated Alzheimer-type phenotype in transgenic mice carrying both mutant amyloid precursor protein and presenilin 1 transgenes

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7 January 2021

1 reference

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7 January 2021

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

1 reference

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7 January 2021

APPSw transgenic mice develop age-related A beta deposits and neuropil abnormalities, but no neuronal loss in CA1.

1 reference

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7 January 2021

Amyloid precursor protein processing and A beta42 deposition in a transgenic mouse model of Alzheimer disease

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7 January 2021

Deposition of beta/A4 immunoreactivity and neuronal pathology in transgenic mice expressing the carboxyl-terminal fragment of the Alzheimer amyloid precursor in the brain

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7 January 2021

The Alzheimer's A beta peptide induces neurodegeneration and apoptotic cell death in transgenic mice

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7 January 2021

Expression in brain of amyloid precursor protein mutated in the alpha-secretase site causes disturbed behavior, neuronal degeneration and premature death in transgenic mice.

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7 January 2021

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7 January 2021

Impaired learning and LTP in mice expressing the carboxy terminus of the Alzheimer amyloid precursor protein.

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7 January 2021

Age-dependent neuronal and synaptic degeneration in mice transgenic for the C terminus of the amyloid precursor protein.

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7 January 2021

Formation of beta-amyloid protein deposits in brains of transgenic mice

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7 January 2021

Amyloid-β Deposition in Alzheimer Transgenic Mice Is Associated with Oxidative Stress

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7 January 2021

Two amyloid precursor protein transgenic mouse models with Alzheimer disease-like pathology

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7 January 2021

An increased percentage of long amyloid beta protein secreted by familial amyloid beta protein precursor (beta APP717) mutants.

1 reference

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7 January 2021

Amyloidogenic role of cytokine TGF-beta1 in transgenic mice and in Alzheimer's disease

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7 January 2021

Increased susceptibility to ischemic brain damage in transgenic mice overexpressing the amyloid precursor protein.

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7 January 2021

10.1016/S0531-5565(98)00045-X

Identifiers

DOI

1 reference

10 February 2018

release_s3i26ykqjvbaxi263w26mi2q7u

Fatcat ID

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PubMed ID

9951631

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10 February 2018