(Q48300905)

10 February 2018

title

Deletion of Nrf2 Exacerbates Oxidative Stress After Traumatic Brain Injury in Mice. (English)

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10 February 2018

author name string

Xin-Yu Lu

1

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10 February 2018

Han-Dong Wang

2

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10 February 2018

Jian-Guo Xu

3

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10 February 2018

Ke Ding

4

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10 February 2018

Tao Li

5

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10 February 2018

publication date

3 March 2015

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10 February 2018

Cellular and Molecular Neurobiology

published in

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10 February 2018

volume

35

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10 February 2018

issue

5

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10 February 2018

page(s)

713-721

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10 February 2018

https://scigraph.springernature.com/pub.10.1007/s10571-015-0167-9

exact match

0 references

cites work

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12 December 2020

Treatment with exogenous hydrogen sulfide attenuates hyperoxia-induced acute lung injury in mice

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12 December 2020

Disruption of Nrf2 enhances upregulation of nuclear factor-kappaB activity, proinflammatory cytokines, and intercellular adhesion molecule-1 in the brain after traumatic brain injury

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12 December 2020

Neuroprotective effects of caffeic acid phenethyl ester on experimental traumatic brain injury in rats

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12 December 2020

Transcription factor Nrf2 protects the spinal cord from inflammation produced by spinal cord injury

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12 December 2020

Attenuation of brain edema and spatial learning deﬁcits by the inhibition of NADPH oxidase activity using apocynin following diffuse traumatic brain injury in rats

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12 December 2020

Activation of Nrf2-ARE pathway in brain after traumatic brain injury

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12 December 2020

Disruption of Nrf2 enhances the upregulation of nuclear factor-kappaB activity, tumor necrosis factor-α, and matrix metalloproteinase-9 after spinal cord injury in mice

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12 December 2020

Malondialdehyde determination as index of lipid peroxidation

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12 December 2020

Variation in the activities of glutathione peroxidase and superoxide dismutase and in the concentration of copper in the blood in various breed crosses of sheep

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12 December 2020

10.1007/S10571-015-0167-9

Identifiers

DOI

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10 February 2018

PubMed ID

25732597

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10 February 2018