(Q48400092)

11 February 2018

title

Role of c-fos in hypoxia-induced AP-1 cis-element activity and tyrosine hydroxylase gene expression (English)

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11 February 2018

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4

Cherniack NS

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11 February 2018

author name string

Mishra RR

1

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11 February 2018

Adhikary G

2

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11 February 2018

Simonson MS

3

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11 February 2018

Prabhakar NR

5

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11 February 2018

publication date

1 August 1998

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11 February 2018

published in

Molecular Brain Research

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11 February 2018

volume

59

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11 February 2018

issue

1

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11 February 2018

page(s)

74-83

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11 February 2018

Regulation of c-jun expression during hypoxic and low-glucose stress

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The N-methyl-D-aspartate receptor pathway is involved in hypoxia-induced c-Fos protein expression in the rat nucleus of the solitary tract

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Hypoxia-inducible factor 1 levels vary exponentially over a physiologically relevant range of O2 tension

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The regulation of AP-1 activity by mitogen-activated protein kinases

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Hypoxia induces endothelin gene expression and secretion in cultured human endothelium

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Hypoxic induction of human vascular endothelial growth factor expression through c-Src activation

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Hypoxia-induced protein binding to O2-responsive sequences on the tyrosine hydroxylase gene

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Cell selective induction and transcriptional activation of immediate early genes by hypoxia

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Difference in transcriptional regulatory function between c-Fos and Fra-2.

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Delayed expression of c-fos protein in rat hippocampus and cerebral cortex following transient in vivo exposure to hypoxia

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7 January 2021

Activation of AP-1 and of a nuclear redox factor, Ref-1, in the response of HT29 colon cancer cells to hypoxia

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7 January 2021

10.1016/S0169-328X(98)00139-9

Identifiers

DOI

1 reference

11 February 2018

1 reference

11 February 2018