(Q48414017)

English

Morphofunctional studies on the neuropeptide Y/adrenaline costoring terminal systems in the dorsal cardiovascular region of the medulla oblongata. Focus on receptor-receptor interactions in cotransmission.

scientific article published in January 1986

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scholarly article

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9 October 2019

Morphofunctional studies on the neuropeptide Y/adrenaline costoring terminal systems in the dorsal cardiovascular region of the medulla oblongata. Focus on receptor-receptor interactions in cotransmission (English)

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9 October 2019

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Fuxe K

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11 February 2018

1

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K Fuxe

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9 October 2019

author name string

L F Agnati

2

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9 October 2019

A Härfstrand

3

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9 October 2019

A M Janson

4

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9 October 2019

A Neumeyer

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9 October 2019

K Andersson

6

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9 October 2019

M Ruggeri

7

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9 October 2019

M Zoli

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9 October 2019

M Goldstein

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9 October 2019

publication date

1 January 1986

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9 October 2019

Progress in Brain Research

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9 October 2019

68

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9 October 2019

303-320

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9 October 2019

Subcortical limbic 3H-N-propylnorapomorphine binding sites are markedly modulated by cholecystokinin-8 in vitro

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New concepts on the structure of the neuronal networks: the miniaturization and hierarchical organization of the central nervous system. (Hypothesis).

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Failure of neuropeptide Y in vitro to increase the number of α2-adrenergic binding sites in membranes of medulla oblongata of the spontaneous hypertensive rat*

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Neurotensin in vitro markedly reduces the affinity in subcortical limbic 3H-N-propylnorapomorphine binding sites.

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Neuropeptide Y in vitro selectively increases the number of α2-adrenergic binding sites in membranes of the medulla oblongata of the rat

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Reduction of adrenaline turnover in cardiovascular areas of rat medulla oblongata by clonidine

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Cholecystokinin peptides produce marked reduction of dopamine turnover in discrete areas in the rat brain following intraventricular injection

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Modulation by cholecystokinins of3H-spiroperidol binding in rat striatum: evidence for increased affinity and reduction in the number of binding sites

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Selective reduction of adrenaline turnover in the dorsal midline area of the caudal medulla oblongata and increase of hypothalamic adrenaline levels in the Lyon strain of genetically hypertensive rats.

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Central administration of neuropeptide Y induces hypotension bradypnea and EEG synchronization in the rat

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Quantitative autoradiographic localization of [3H]imipramine binding sites in the brain of the rat: relationship to ascending 5-hydroxytryptamine neuron systems

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Evidence for a modulation by neuropeptide Y of the alpha-2 adrenergic transmission line in central adrenaline synapses. New possibilities for treatment of hypertensive disorders.

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Studies on neurotensin catecholamine interactions in the hypothalamus and in the forebrain of the male rat

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Medianosomes as integrative units in the external layer of the median eminence. Studies on grf/catecholamine and somatostatin/catecholamine interactions in the hypothalamus of the male rat

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Studies on neuropeptide-Y catecholamine interactions in central cardiovascular regulation in the alpha-chloralose anaesthetized rat. Evidence for a possible new way of activating the alpha-2 adrenergic transmission line.

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Glucocorticoid receptor immunoreactivity in monoaminergic neurons of rat brain

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Transmitter and peptide systems in areas involved in the control of blood pressure

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7 January 2021

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Distinct cholecystokinin receptors in brain and pancreas

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Rat medulla oblongata. IV. Topographical distribution of catecholaminergic neurons with quantitative three-dimensional computer reconstruction

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Regulation of neuropeptide Y (NPY) binding by guanine nucleotides in the rat cerebral cortex.

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Actions of centrally administered neuropeptide Y on EEG activity in different rat strains and in different phases of their circadian cycle.

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Identifiers

10.1016/S0079-6123(08)60246-0

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:3031731%20AND%20SRC:MED&resulttype=core&format=json

9 October 2019

PubMed publication ID

1 reference

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9 October 2019

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