(Q48516634)

12 February 2018

title

Gender-specific neuroprotection by 2-iminobiotin after hypoxia-ischemia in the neonatal rat via a nitric oxide independent pathway. (English)

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12 February 2018

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2

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12 February 2018

Henrik Hagberg

4

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12 February 2018

Cobi J Heijnen

6

object named as

Cobi J Heijnen

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12 February 2018

Annemieke Kavelaars

3

object named as

Annemieke Kavelaars

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12 February 2018

Cora H Nijboer

1

object named as

Cora H A Nijboer

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12 February 2018

author name string

Frank van Bel

5

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12 February 2018

publication date

31 May 2006

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12 February 2018

Journal of Cerebral Blood Flow & Metabolism

published in

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12 February 2018

volume

27

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12 February 2018

issue

2

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12 February 2018

page(s)

282-292

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12 February 2018

Gender-linked brain injury in experimental stroke.

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Developmental changes in the sensitivity of the neonatal rat brain to hypoxic/ischemic injury

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Adult or perinatal brain injury: does sex matter?

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Ischemic nitric oxide and poly (ADP-ribose) polymerase-1 in cerebral ischemia: male toxicity, female protection

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Apoptosis in perinatal hypoxic-ischemic brain injury: how important is it and should it be inhibited?

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Inducible nitric oxide synthase contributes to gender differences in ischemic brain injury

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Neuroprotection by selective nitric oxide synthase inhibition at 24 hours after perinatal hypoxia-ischemia

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The influence of immaturity on hypoxic‐ischemic brain damage in the rat

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At what age is the developing cerebral cortex of the rat comparable to that of the full-term newborn human baby?

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2-Iminobiotin is an inhibitor of nitric oxide synthases

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Estrogen-mediated neuroprotection after experimental stroke in male rats

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Inhibition of nNOS and iNOS following hypoxia-ischaemia improves long-term outcome but does not influence the inflammatory response in the neonatal rat brain

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Long-term neuroprotection with 2-iminobiotin, an inhibitor of neuronal and inducible nitric oxide synthase, after cerebral hypoxia-ischemia in neonatal rats

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Bilateral molecular changes in a neonatal rat model of unilateral hypoxic-ischemic brain damage.

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7 January 2021

Expression of nitric oxide synthase isoforms and nitrotyrosine formation after hypoxia-ischemia in the neonatal rat brain

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Apoptosis-inducing factor substitutes for caspase executioners in NMDA-triggered excitotoxic neuronal death.

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7 January 2021

The effect of age on susceptibility to hypoxic-ischemic brain damage

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Mediation of poly(ADP-ribose) polymerase-1-dependent cell death by apoptosis-inducing factor

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Nitrosylation precedes caspase-3 activation and translocation of apoptosis-inducing factor in neonatal rat cerebral hypoxia-ischaemia

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The influence of age on apoptotic and other mechanisms of cell death after cerebral hypoxia-ischemia.

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7 January 2021

Different apoptotic mechanisms are activated in male and female brains after neonatal hypoxia-ischaemia

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7 January 2021

Identifiers

DOI

10.1038/SJ.JCBFM.9600342

1 reference

12 February 2018

PubMed ID

16736041

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12 February 2018