(Q48573667)

12 February 2018

title

Thalamic neuron apoptosis emerges rapidly after cortical damage in immature mice. (English)

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12 February 2018

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Natale JE

1

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12 February 2018

Cheng Y

series ordinal

2

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12 February 2018

Martin LJ

series ordinal

3

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12 February 2018

language of work or name

English

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publication date

1 January 2002

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12 February 2018

published in

Neuroscience

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12 February 2018

volume

112

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12 February 2018

issue

3

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12 February 2018

page(s)

665-676

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12 February 2018

Axotomy-induced neuronal death and reactive astrogliosis in the lateral geniculate nucleus following a lesion of the visual cortex in the rat.

cites work

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Apoptosis of retrogradely degenerating neurons occurs in association with the accumulation of perikaryal mitochondria and oxidative damage to the nucleus

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Occipital cortex ablation in adult rat causes retrograde neuronal death in the lateral geniculate nucleus that resembles apoptosis.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Ultrastructure of retrograde degeneration in thalamus of rat. 1. Neuronal somata and dendrites.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Apoptotic neurodegeneration following trauma is markedly enhanced in the immature brain.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Caspase-3 mediated neuronal death after traumatic brain injury in rats

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

A biochemical hallmark of apoptosis: internucleosomal degradation of the genome

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Experimental brain injury induces regionally distinct apoptosis during the acute and delayed post-traumatic period.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Analysis of the mechanism of loss of trophic factor dependence associated with neuronal maturation: a phenotype indistinguishable from Bax deletion.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

The role of excitatory amino acids and NMDA receptors in traumatic brain injury

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

An autoradiographic study of the development of the entorhinal and commissural afferents to the dentate gyrus of the rat

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Disruption of epithelial cell-matrix interactions induces apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Neural damage in the rat thalamus after cortical infarcts

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

The nucleolar apparatus and neuronal reactivity to injury during development

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Neurodegeneration in excitotoxicity, global cerebral ischemia, and target deprivation: A perspective on the contributions of apoptosis and necrosis.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Injury-induced apoptosis of neurons in adult brain is mediated by p53-dependent and p53-independent pathways and requires Bax.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

The extracellular matrix as a cell survival factor

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Distribution and retrograde transport of trophic factors in the central nervous system: functional implications for the treatment of neurodegenerative diseases

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Apoptosis has a prolonged role in the neurodegeneration after hypoxic ischemia in the newborn rat.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Temporal profile of apoptotic-like changes in neurons and astrocytes following controlled cortical impact injury in the rat.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Delayed neurodegeneration in neonatal rat thalamus after hypoxia-ischemia is apoptosis.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Cell death during development of the nervous system

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

The postnatal development of neurons in the dorsal lateral geniculate nucleus of the rat: a Golgi study.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

A system for characterizing cellular and molecular events in programmed neuronal cell death.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

N-Methyl-D-aspartate antagonists and apoptotic cell death triggered by head trauma in developing rat brain

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Thalamic retrograde degeneration following cortical injury: an excitotoxic process?

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Anchorage dependence, integrins, and apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Fluoro-Gold: a new fluorescent retrograde axonal tracer with numerous unique properties.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

Activation of CPP32-like caspases contributes to neuronal apoptosis and neurological dysfunction after traumatic brain injury.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2802%2900098-2

7 January 2021

10.1016/S0306-4522(02)00098-2

Identifiers

DOI

1 reference

12 February 2018

1 reference

12 February 2018