(Q48636627)

English

The neuronal migration defect in mice with Zellweger syndrome (Pex5 knockout) is not caused by the inactivity of peroxisomal beta-oxidation.

scientific article published in April 2002

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scholarly article

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

The neuronal migration defect in mice with Zellweger syndrome (Pex5 knockout) is not caused by the inactivity of peroxisomal beta-oxidation. (English)

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

Zellweger syndrome

1 reference

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inferred from title

Pierre Gressens

2

object named as

P Gressens

1 reference

Europe PubMed Central

13 February 2018

author name string

M Baes

1

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

S Huyghe

3

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

Nys K De

4

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

C Qi

5

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

Y Jia

6

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

G P Mannaerts

7

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

P Evrard

8

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

Veldhoven P P Van

9

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

P E Declercq

10

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

J K Reddy

11

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

publication date

1 April 2002

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

Journal of Neuropathology & Experimental Neurology

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Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

61

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Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

4

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

368-374

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

Plasma very long chain fatty acids in 3,000 peroxisome disease patients and 29,000 controls

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

Polyunsaturated fatty acids in the developing human brain, erythrocytes and plasma in peroxisomal disease: therapeutic implications

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

Neuronal migration abnormality in peroxisomal bifunctional enzyme defect.

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

Interactions of a very long chain fatty acid with model membranes and serum albumin. Implications for the pathogenesis of adrenoleukodystrophy

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

A mouse model for Zellweger syndrome

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

Targeted deletion of the PEX2 peroxisome assembly gene in mice provides a model for Zellweger syndrome, a human neuronal migration disorder

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

Peroxisomal lipid degradation via beta- and alpha-oxidation in mammals

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

Absence of spontaneous peroxisome proliferation in enoyl-CoA Hydratase/L-3-hydroxyacyl-CoA dehydrogenase-deficient mouse liver. Further support for the role of fatty acyl CoA oxidase in PPARalpha ligand metabolism

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

The deficient degradation of synthetic 2- and 3-methyl-branched fatty acids in fibroblasts from patients with peroxisomal disorders

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

Docosahexaenoic acid deficit is not a major pathogenic factor in peroxisome-deficient mice

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

Targeted inactivation of the X-linked adrenoleukodystrophy gene in mice

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

A mouse model for X-linked adrenoleukodystrophy

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

Adrenoleukodystrophy protein-deficient mice represent abnormality of very long chain fatty acid metabolism

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

Hepatocellular and hepatic peroxisomal alterations in mice with a disrupted peroxisomal fatty acyl-coenzyme A oxidase gene

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

Abnormal profiles of polyunsaturated fatty acids in the brain, liver, kidney and retina of patients with peroxisomal disorders

1 reference

https://api.crossref.org/works/10.1093%2FJNEN%2F61.4.368

21 January 2018

Identifiers

10.1093/JNEN/61.4.368

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

release_vljkf4cmvnhabnlz5momk3zuka

1 reference

https://api.fatcat.wiki/v0/release/vljkf4cmvnhabnlz5momk3zuka

24 November 2022

based on heuristic

mapped directly with Wikidata item

1 reference

Europe PubMed Central

13 February 2018

http://europepmc.org/abstract/MED/11939592

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