(Q48697807)

14 February 2018

title

Constitutive expression of costimulatory molecules by human microglia and its relevance to CNS autoimmunity. (English)

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14 February 2018

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4

Hafler DA

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14 February 2018

author name string

Dangond F

series ordinal

1

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14 February 2018

Windhagen A

series ordinal

2

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14 February 2018

Groves CJ

series ordinal

3

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14 February 2018

publication date

1 June 1997

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14 February 2018

Journal of Neuroimmunology

published in

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14 February 2018

volume

76

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14 February 2018

issue

1-2

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14 February 2018

page(s)

132-138

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14 February 2018

Functional expression of B7/BB1 on activated T lymphocytes

cites work

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Microglia present myelin antigens to T cells after phagocytosis of oligodendrocytes

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B70/B7-2 is identical to CD86 and is the major functional ligand for CD28 expressed on human dendritic cells

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The costimulatory molecule B7 is expressed on human microglia in culture and in multiple sclerosis acute lesions.

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B7-1 and B7-2 do not deliver identical costimulatory signals, since B7-2 but not B7-1 preferentially costimulates the initial production of IL-4.

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B7-1 and B7-2 costimulatory molecules activate differentially the Th1/Th2 developmental pathways: application to autoimmune disease therapy

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Differential effects of anti-B7-1 and anti-B7-2 monoclonal antibody treatment on the development of diabetes in the nonobese diabetic mouse

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Treatment of multiple sclerosis with gamma interferon: exacerbations associated with activation of the immune system

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Distinct roles for B7-1 (CD-80) and B7-2 (CD-86) in the initiation of experimental allergic encephalomyelitis

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T-cell costimulatory molecules B7-1 (CD80) and B7-2 (CD86) are expressed in human microglia but not in astrocytes in culture

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T cells involved in psoriasis vulgaris belong to the Th1 subset

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Costimulation of T lymphocytes: the role of CD28, CTLA-4, and B7/BB1 in interleukin-2 production and immunotherapy

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Expression of costimulatory molecules B7-1 (CD80), B7-2 (CD86), and interleukin 12 cytokine in multiple sclerosis lesions

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Identifiers

DOI

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14 February 2018

1 reference

14 February 2018