(Q48713760)

English

Free radical spin trap alpha-phenyl-N-tert-butyl-nitron inhibits caspase-3 activation and reduces brain damage following a severe forebrain ischemic injury.

scientific article published in November 2001

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scholarly article

1 reference

Europe PubMed Central

14 February 2018

http://europepmc.org/abstract/MED/11705697

Free radical spin trap alpha-phenyl-N-tert-butyl-nitron inhibits caspase-3 activation and reduces brain damage following a severe forebrain ischemic injury. (English)

1 reference

Europe PubMed Central

14 February 2018

http://europepmc.org/abstract/MED/11705697

author name string

Li PA

1

1 reference

Europe PubMed Central

14 February 2018

http://europepmc.org/abstract/MED/11705697

He QP

2

1 reference

Europe PubMed Central

14 February 2018

http://europepmc.org/abstract/MED/11705697

Nakamura L

3

1 reference

Europe PubMed Central

14 February 2018

http://europepmc.org/abstract/MED/11705697

Csiszar K

4

1 reference

Europe PubMed Central

14 February 2018

http://europepmc.org/abstract/MED/11705697

publication date

1 November 2001

1 reference

Europe PubMed Central

14 February 2018

http://europepmc.org/abstract/MED/11705697

Free Radical Biology and Medicine

1 reference

Europe PubMed Central

14 February 2018

http://europepmc.org/abstract/MED/11705697

31

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Europe PubMed Central

14 February 2018

http://europepmc.org/abstract/MED/11705697

10

1 reference

Europe PubMed Central

14 February 2018

http://europepmc.org/abstract/MED/11705697

1191-1197

1 reference

Europe PubMed Central

14 February 2018

http://europepmc.org/abstract/MED/11705697

Free radical damage to protein and DNA: mechanisms involved and relevant observations on brain undergoing oxidative stress

1 reference

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Reactive oxygen species and the central nervous system

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alpha-Phenyl-tert-butyl-nitrone reduces cortical infarct and edema in rats subjected to focal ischemia.

1 reference

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7 January 2021

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Delayed treatment with alpha-phenyl-N-tert-butyl nitrone (PBN) attenuates secondary mitochondrial dysfunction after transient focal cerebral ischemia in the rat.

1 reference

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Neuroprotective effects of a novel nitrone, NXY-059, after transient focal cerebral ischemia in the rat.

1 reference

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7 January 2021

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NXY-059, a free radical--trapping agent, substantially lessens the functional disability resulting from cerebral ischemia in a primate species

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Delayed treatment with the spin trap alpha-phenyl-N-tert-butyl nitrone (PBN) reduces infarct size following transient middle cerebral artery occlusion in rats

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N-tert-butyl-alpha-phenylnitrone improves recovery of brain energy state in rats following transient focal ischemia

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Increased oxidative stress brought on by pro-inflammatory cytokines in neurodegenerative processes and the protective role of nitrone-based free radical traps

1 reference

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7 January 2021

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Inhibition of NF-kappaB, iNOS mRNA, COX2 mRNA, and COX catalytic activity by phenyl-N-tert-butylnitrone (PBN).

1 reference

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Oxygen free radical involvement in ischemia and reperfusion injury to brain

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Effects of the spin trap-alpha-phenyl-N-tert-butyl nitrone (PBN) in transient forebrain ischaemia in the rat

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The mitochondrial death/life regulator in apoptosis and necrosis

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Models for studying long-term recovery following forebrain ischemia in the rat. 2. A 2-vessel occlusion model.

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Effects of intracarotid arterial injection of cyclosporin A and spontaneous hypothermia on brain damage incurred after a long period of global ischemia

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Cyclosporin A enhances survival, ameliorates brain damage, and prevents secondary mitochondrial dysfunction after a 30-minute period of transient cerebral ischemia

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Influence of in vitro lactic acidosis and hypercapnia on respiratory activity of isolated rat brain mitochondria.

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Lactic acidosis and recovery of mitochondrial function following forebrain ischemia in the rat.

1 reference

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Recovery of brain mitochondrial function in the rat after complete and incomplete cerebral ischemia

1 reference

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Mitochondrial response to transient forebrain ischemia and recirculation in the rat.

1 reference

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7 January 2021

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Altered mitochondrial respiration in selectively vulnerable brain subregions following transient forebrain ischemia in the rat.

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Effect of reperfusion following cerebral ischaemia on the activity of the mitochondrial respiratory chain in the gerbil brain.

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Mitochondrial calcium transport: physiological and pathological relevance

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The mitochondrial permeability transition.

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Recent progress on regulation of the mitochondrial permeability transition pore; a cyclosporin-sensitive pore in the inner mitochondrial membrane

1 reference

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The permeability transition pore. Control points of a cyclosporin A-sensitive mitochondrial channel involved in cell death

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The apoptosis-necrosis paradox. Apoptogenic proteases activated after mitochondrial permeability transition determine the mode of cell death.

1 reference

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Mitochondrial permeability transition in apoptosis and necrosis

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Mitochondrial control of nuclear apoptosis.

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Mitochondrial implication in accidental and programmed cell death: apoptosis and necrosis

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Induction of caspase-3-like protease may mediate delayed neuronal death in the hippocampus after transient cerebral ischemia.

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Activation and cleavage of caspase-3 in apoptosis induced by experimental cerebral ischemia.

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Attenuation of delayed neuronal death after mild focal ischemia in mice by inhibition of the caspase family.

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Caspase inhibition reduces myocyte cell death induced by myocardial ischemia and reperfusion in vivo

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Identifiers

10.1016/S0891-5849(01)00700-6

1 reference

Europe PubMed Central

14 February 2018

http://europepmc.org/abstract/MED/11705697

1 reference

Europe PubMed Central

14 February 2018

http://europepmc.org/abstract/MED/11705697

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