(Q48811571)

15 February 2018

title

Mitochondria accumulate Ca2+ following intense glutamate stimulation of cultured rat forebrain neurones. (English)

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15 February 2018

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R J White

1

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15 February 2018

I J Reynolds

series ordinal

2

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15 February 2018

language of work or name

English

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publication date

1 January 1997

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15 February 2018

published in

Journal of Physiology

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15 February 2018

volume

498 ( Pt 1)

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15 February 2018

page(s)

31-47

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15 February 2018

Calcium Dependence of Toxic Cell Death: A Final Common Pathway

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12 December 2020

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NGF and bFGF protect rat hippocampal and human cortical neurons against hypoglycemic damage by stabilizing calcium homeostasis

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Glutamate-induced calcium transient triggers delayed calcium overload and neurotoxicity in rat hippocampal neurons

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A role for the mitochondrial Na(+)-Ca2+ exchanger in the regulation of oxidative phosphorylation in isolated heart mitochondria

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Calcium homeostasis in rat septal neurons in tissue culture

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Selectivity of inhibition of Na(+)-Ca2+ exchange of heart mitochondria by benzothiazepine CGP-37157

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Glutamate receptor-induced 45Ca2+ accumulation in cortical cell culture correlates with subsequent neuronal degeneration.

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Source specificity of early calcium neurotoxicity in cultured embryonic spinal neurons

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Changes in intracellular pH associated with glutamate excitotoxicity

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Microdomains with high Ca2+ close to IP3-sensitive channels that are sensed by neighboring mitochondria

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Traumatic brain injury-induced excitotoxicity assessed in a controlled cortical impact model

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Nitric oxide activation of poly(ADP-ribose) synthetase in neurotoxicity

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The quantity of calcium that appears to induce neuronal death.

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N-methyl-D-aspartate induces a rapid, reversible, and calcium-dependent intracellular acidosis in cultured fetal rat hippocampal neurons

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Perturbation of intracellular calcium and hydrogen ion regulation in cultured mouse hippocampal neurons by reduction of the sodium ion concentration gradient.

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Mitochondrial calcium transport: physiological and pathological relevance

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Mechanisms of glutamate-stimulated Mg2+ influx and subsequent Mg2+ efflux in rat forebrain neurones in culture

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Mitochondrial depolarization in glutamate-stimulated neurons: an early signal specific to excitotoxin exposure

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Mitochondrial Ca2+ transport and the role of intramitochondrial Ca2+ in the regulation of energy metabolism

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The sodium-calcium antiport of heart mitochondria is not electroneutral

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Glutamate-induced destabilization of intracellular calcium concentration homeostasis in cultured cerebellar granule cells: role of mitochondria in calcium buffering

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Inhibition of the mitochondrial Ca2+ uniporter by pure and impure ruthenium red.

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Mitochondria and Na+/Ca2+ exchange buffer glutamate-induced calcium loads in cultured cortical neurons

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Recent progress on regulation of the mitochondrial permeability transition pore; a cyclosporin-sensitive pore in the inner mitochondrial membrane

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Glutamate-induced intracellular acidification of cultured hippocampal neurons demonstrates altered energy metabolism resulting from Ca2+ loads

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Calcium: still center-stage in hypoxic-ischemic neuronal death

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Regulation of intracellular calcium and calcium buffering properties of rat isolated neurohypophysial nerve endings

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Glutamate induces the production of reactive oxygen species in cultured forebrain neurons following NMDA receptor activation

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12 December 2020

Intracellular acidification is not a prerequisite for glutamate-triggered death of cultured hippocampal neurons

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Free radicals, calcium homeostasis, heat shock proteins, and myocardial stunning

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The role of hydroperoxides as calcium release agents in rat brain mitochondria.

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10.1113/JPHYSIOL.1997.SP021839

Identifiers

DOI

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15 February 2018

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15 February 2018