(Q49090197)

English

Impairment of mitochondrial respiration after cerebral hypoxia-ischemia in immature rats: relationship to activation of caspase-3 and neuronal injury.

scientific article published in December 2000

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

Impairment of mitochondrial respiration after cerebral hypoxia-ischemia in immature rats: relationship to activation of caspase-3 and neuronal injury. (English)

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

0 references

8

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

9

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

Puka-Sundvall M

1

object named as

Puka-Sundvall M

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

author name string

Wallin C

2

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

Gilland E

3

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

Hallin U

4

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

Wang X

5

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

Sandberg M

6

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

Karlsson J

7

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

publication date

1 December 2000

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

number of pages

8

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inferred from page(s)

Developmental Brain Research

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

125

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

1-2

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

43-50

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

Effect of reperfusion following cerebral ischaemia on the activity of the mitochondrial respiratory chain in the gerbil brain.

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Relation between delayed impairment of cerebral energy metabolism and infarction following transient focal hypoxia-ischaemia in the developing brain

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The mitochondrial permeability transition pore and its role in cell death

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Differences in the activation of the mitochondrial permeability transition among brain regions in the rat correlate with selective vulnerability.

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Temporal changes of regional glucose use, blood flow, and microtubule-associated protein 2 immunostaining after hypoxia-ischemia in the immature rat brain.

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NMDA Receptor-dependent increase of cerebral glucose utilization after hypoxia-ischemia in the immature rat.

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Mitochondrial function and energy metabolism after hypoxia-ischemia in the immature rat brain: involvement of NMDA-receptors.

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Molecular characterization of microtubule-associated proteins tau and MAP2.

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Mitochondria and apoptosis

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Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion

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Hypoxia-ischemia in the neonatal rat brain: histopathology after post-treatment with NMDA and non-NMDA receptor antagonists

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Inhibition of interleukin 1beta converting enzyme family proteases reduces ischemic and excitotoxic neuronal damage

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Mitochondrial response to transient forebrain ischemia and recirculation in the rat.

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Regional Alterations of ATP and Heat-Shock Protein-72 mRNA following Hypoxia—Ischemia in Neonatal Rat Brain

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The mitochondrial death/life regulator in apoptosis and necrosis

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Secondary bioenergetic failure after transient focal ischaemia is due to mitochondrial injury.

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An ICE inhibitor, z-VAD-DCB attenuates ischaemic brain damage in the rat.

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Delayed ("secondary") cerebral energy failure after acute hypoxia-ischemia in the newborn piglet: continuous 48-hour studies by phosphorus magnetic resonance spectroscopy

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Identifiers

10.1016/S0165-3806(00)00111-5

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11154759

ResearchGate publication ID

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