(Q50511873)

English

Inhibition of toxicity in the beta-amyloid peptide fragment beta -(25-35) using N-methylated derivatives: a general strategy to prevent amyloid formation.

scientific article published in August 2000

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

14 March 2018

http://europepmc.org/abstract/MED/10825171

Inhibition of toxicity in the beta-amyloid peptide fragment beta -(25-35) using N-methylated derivatives: a general strategy to prevent amyloid formation. (English)

1 reference

Europe PubMed Central

PubMed publication ID

14 March 2018

http://europepmc.org/abstract/MED/10825171

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3

1 reference

Europe PubMed Central

PubMed publication ID

14 March 2018

http://europepmc.org/abstract/MED/10825171

author name string

E Hughes

1

1 reference

Europe PubMed Central

PubMed publication ID

14 March 2018

http://europepmc.org/abstract/MED/10825171

R M Burke

2

1 reference

Europe PubMed Central

PubMed publication ID

14 March 2018

http://europepmc.org/abstract/MED/10825171

language of work or name

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publication date

1 August 2000

1 reference

Europe PubMed Central

PubMed publication ID

14 March 2018

http://europepmc.org/abstract/MED/10825171

Journal of Biological Chemistry

1 reference

Europe PubMed Central

PubMed publication ID

14 March 2018

http://europepmc.org/abstract/MED/10825171

275

1 reference

Europe PubMed Central

PubMed publication ID

14 March 2018

http://europepmc.org/abstract/MED/10825171

33

1 reference

Europe PubMed Central

PubMed publication ID

14 March 2018

http://europepmc.org/abstract/MED/10825171

25109-25115

1 reference

Europe PubMed Central

PubMed publication ID

14 March 2018

http://europepmc.org/abstract/MED/10825171

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

The toxicity in vitro of beta-amyloid protein

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Normal and abnormal biology of the beta-amyloid precursor protein

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Amyloid beta-protein and the genetics of Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

X-ray diffraction from intraneuronal paired helical filaments and extraneuronal amyloid fibers in Alzheimer disease indicates cross-beta conformation

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Reversible random coil-beta-sheet transition of the Alzheimer beta-amyloid fragment (25-35).

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Inhibition of PC12 cell redox activity is a specific, early indicator of the mechanism of beta-amyloid-mediated cell death

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Vitamin E protects nerve cells from amyloid beta protein toxicity

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Role of the beta-amyloid precursor protein in Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

A beta A4 amyloid precursor protein gene and Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Cloning of a gene bearing missense mutations in early-onset familial Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Familial Alzheimer's disease in kindreds with missense mutations in a gene on chromosome 1 related to the Alzheimer's disease type 3 gene

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

The presenilin 2 mutation (N141I) linked to familial Alzheimer disease (Volga German families) increases the secretion of amyloid beta protein ending at the 42nd (or 43rd) residue

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Assembly of A beta amyloid protofibrils: an in vitro model for a possible early event in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Beta-amyloid neurotoxicity requires fibril formation and is inhibited by congo red.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Inhibition of amyloid formation: a strategy to delay the onset of Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Two-hybrid system as a model to study the interaction of beta-amyloid peptide monomers

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Beta-sheet breaker peptides inhibit fibrillogenesis in a rat brain model of amyloidosis: implications for Alzheimer's therapy

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Quantitative evaluation of congo red binding to amyloid-like proteins with a beta-pleated sheet conformation

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Physical, morphological and functional differences between ph 5.8 and 7.4 aggregates of the Alzheimer's amyloid peptide Abeta

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Thioflavine T interaction with synthetic Alzheimer's disease beta-amyloid peptides: detection of amyloid aggregation in solution

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Re-examination and further development of a precise and rapid dye method for measuring cell growth/cell kill

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Untangling the pathophysio-chemistry of β-amyloid

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Controlling amyloid beta-peptide fibril formation with protease-stable ligands

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Modified-peptide inhibitors of amyloid beta-peptide polymerization

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Conformational changes and disease--serpins, prions and Alzheimer's.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Protein misfolding, evolution and disease

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Aberrant protein deposition and neurological disease

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

All-D-enantiomers of beta-amyloid exhibit similar biological properties to all-L-beta-amyloids.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

A strategy for designing inhibitors of beta-amyloid toxicity

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Arrest of beta-amyloid fibril formation by a pentapeptide ligand

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Inhibition of Alzheimer's amyloidosis by peptides that prevent beta-sheet conformation

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Specific inhibition of in vitro formation of protease-resistant prion protein by synthetic peptides

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Reversion of prion protein conformational changes by synthetic b-sheet breaker peptides

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Conformational energy studies on N-methylated analogs of thyrotropin releasing hormone, enkephalin, and luteinizing hormone-releasing hormone

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Prolines and amyloidogenicity in fragments of the Alzheimer's peptide beta/A4

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

The intracellular component of cellular 3-(4,5-dimethylthiazol-2-yl)-2, 5-diphenyltetrazolium bromide (MTT) reduction is specifically inhibited by beta-amyloid peptides.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M003554200

21 January 2018

Identifiers

10.1074/JBC.M003554200

1 reference

Europe PubMed Central

PubMed publication ID

14 March 2018

http://europepmc.org/abstract/MED/10825171

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

14 March 2018

http://europepmc.org/abstract/MED/10825171

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