(Q53333352)

13 May 2018

review article

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title

Conformational changes and disease--serpins, prions and Alzheimer's. (English)

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13 May 2018

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2

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13 May 2018

author name string

Carrell RW

series ordinal

1

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13 May 2018

publication date

1 December 1998

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13 May 2018

Current Opinion in Structural Biology

published in

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13 May 2018

volume

8

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13 May 2018

issue

6

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13 May 2018

page(s)

799-809

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13 May 2018

cites work

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The anticoagulant activation of antithrombin by heparin

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Structural basis of latency in plasminogen activator inhibitor-1

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Autocatalytic processes in cooperative mechanisms of prion diseases

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Aging renders the brain vulnerable to amyloid beta-protein neurotoxicity

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Alzheimer's disease. Tangle disentanglement.

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Association of missense and 5'-splice-site mutations in tau with the inherited dementia FTDP-17

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7 January 2021

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Identification of the binding site for a low-molecular-weight inhibitor of plasminogen activator inhibitor type 1 by site-directed mutagenesis

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7 January 2021

Perlecan binds to the beta-amyloid proteins (A beta) of Alzheimer's disease, accelerates A beta fibril formation, and maintains A beta fibril stability.

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7 January 2021

Haemoglobin Köln (β–98 Valine → Methionine): An Unstable Protein Causing Inclusion-Body Anaemia

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Increased body temperature accelerates aggregation of the Leu-68-->Gln mutant cystatin C, the amyloid-forming protein in hereditary cystatin C amyloid angiopathy.

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Assembly of microtubule-associated protein tau into Alzheimer-like filaments induced by sulphated glycosaminoglycans.

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7 January 2021

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Identifiers

DOI

1 reference

13 May 2018

1 reference

13 May 2018