(Q51007701)

English

Increases in β-amyloid protein in the hippocampus caused by diabetic metabolic disorder are blocked by minocycline through inhibition of NF-κB pathway activation.

scientific article published in March 2011

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

26 March 2018

http://europepmc.org/abstract/MED/21602593

Increases in β-amyloid protein in the hippocampus caused by diabetic metabolic disorder are blocked by minocycline through inhibition of NF-κB pathway activation. (English)

1 reference

Europe PubMed Central

PubMed publication ID

26 March 2018

http://europepmc.org/abstract/MED/21602593

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1 reference

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author name string

Zhiyou Cai

1

1 reference

Europe PubMed Central

PubMed publication ID

26 March 2018

http://europepmc.org/abstract/MED/21602593

Yu Zhao

2

1 reference

Europe PubMed Central

PubMed publication ID

26 March 2018

http://europepmc.org/abstract/MED/21602593

Shengtao Yao

3

1 reference

Europe PubMed Central

PubMed publication ID

26 March 2018

http://europepmc.org/abstract/MED/21602593

Bin Bin Zhao

4

1 reference

Europe PubMed Central

PubMed publication ID

26 March 2018

http://europepmc.org/abstract/MED/21602593

publication date

1 March 2011

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Europe PubMed Central

PubMed publication ID

26 March 2018

http://europepmc.org/abstract/MED/21602593

Pharmacological Reports

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Europe PubMed Central

PubMed publication ID

26 March 2018

http://europepmc.org/abstract/MED/21602593

63

1 reference

Europe PubMed Central

PubMed publication ID

26 March 2018

http://europepmc.org/abstract/MED/21602593

2

1 reference

Europe PubMed Central

PubMed publication ID

26 March 2018

http://europepmc.org/abstract/MED/21602593

381-391

1 reference

Europe PubMed Central

PubMed publication ID

26 March 2018

http://europepmc.org/abstract/MED/21602593

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Amyloid beta-peptide stimulates nitric oxide production in astrocytes through an NFkappaB-dependent mechanism

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Minocycline alleviates hypoxic-ischemic injury to developing oligodendrocytes in the neonatal rat brain.

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Minocycline reduces astrocytic reactivation and neuroinflammation in the hippocampus of a vascular cognitive impairment rat model

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Minocycline attenuates cognitive impairment and restrains oxidative stress in the hippocampus of rats with chronic cerebral hypoperfusion

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The antioxidant effect exerted by TGF-1beta-stimulated hyaluronan production reduced NF-kB activation and apoptosis in human fibroblasts exposed to FeSo4 plus ascorbate

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Minocycline attenuates neuronal cell death and improves cognitive impairment in Alzheimer's disease models

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Oxidative-nitrosative stress and post-translational protein modifications: implications to lung structure-function relations. Arginase modulates NF-kappaB activity via a nitric oxide-dependent mechanism

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Therapeutic rescue of neurodegeneration in experimental type 3 diabetes: Relevance to Alzheimer's disease

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Classification and basic pathology of Alzheimer disease

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Oxidative stress is a critical mediator of the angiotensin II signal in human neutrophils: involvement of mitogen-activated protein kinase, calcineurin, and the transcription factor NF-kappaB.

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Does insulin dysfunction play a role in Alzheimer's disease?

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Beta-amyloid-induced inflammation and cholinergic hypofunction in the rat brain in vivo: involvement of the p38MAPK pathway.

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Overexpression of inducible nitric oxide synthase and cyclooxygenase-2 in rat zinc-deficient lung: Involvement of a NF-kappaB dependent pathway.

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The cross-talk between NF-kappaB and HIF-1: further evidence for a significant liaison

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Gene expression alterations in brain areas of intracerebroventricular streptozotocin treated rat.

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Beta-site amyloid precursor protein cleaving enzyme 1 (BACE1) as a biological candidate marker of Alzheimer's disease

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Delayed treatment with minocycline ameliorates neurologic impairment through activated microglia expressing a high-mobility group box1-inhibiting mechanism

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TNF-alpha-induced NF-kappaB/RelA Ser(276) phosphorylation and enhanceosome formation is mediated by an ROS-dependent PKAc pathway.

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Inhibition of proteasome function leads to NF-κB-independent IL-8 expression in human hepatocytes

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Minocycline and neurodegenerative diseases

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Inhibitory action of minocycline on lipopolysaccharide-induced release of nitric oxide and prostaglandin E2 in BV2 microglial cells

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Attenuation of oxidative stress, inflammation and apoptosis by minocycline prevents retrovirus-induced neurodegeneration in mice

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Inhibitory effect of green tea extract on beta-amyloid-induced PC12 cell death by inhibition of the activation of NF-kappaB and ERK/p38 MAP kinase pathway through antioxidant mechanisms.

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Interleukin-1 beta and tumor necrosis factor-alpha induce expression of alpha 1-antichymotrypsin in human astrocytoma cells by activation of nuclear factor-kappa B.

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Chronic treatment with minocycline preserves adult new neurons and reduces functional impairment after focal cerebral ischemia

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1 reference

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1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

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1 reference

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1 reference

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1 reference

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1 reference

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NF-kappaB and not the MAPK signaling pathway regulates GADD45beta expression during acute inflammation

1 reference

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1 reference

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Identifiers

10.1016/S1734-1140(11)70504-7

1 reference

Europe PubMed Central

PubMed publication ID

26 March 2018

http://europepmc.org/abstract/MED/21602593

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

26 March 2018

http://europepmc.org/abstract/MED/21602593

ResearchGate publication ID

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