(Q51107150)

English

Mixtures of SCFA, composed according to physiologically available concentrations in the gut lumen, modulate histone acetylation in human HT29 colon cancer cells.

scientific article published in November 2006

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scholarly article

1 reference

Europe PubMed Central

29 March 2018

http://europepmc.org/abstract/MED/17092367

Mixtures of SCFA, composed according to physiologically available concentrations in the gut lumen, modulate histone acetylation in human HT29 colon cancer cells. (English)

1 reference

Europe PubMed Central

29 March 2018

http://europepmc.org/abstract/MED/17092367

histone acetylation

1 reference

based on heuristic

inferred from title

1 reference

based on heuristic

inferred from title

Beatrice L. Pool-Zobel

3

object named as

Beatrice L Pool-Zobel

1 reference

Europe PubMed Central

29 March 2018

http://europepmc.org/abstract/MED/17092367

author name string

Jeannette Kiefer

1

1 reference

Europe PubMed Central

29 March 2018

http://europepmc.org/abstract/MED/17092367

Gabriele Beyer-Sehlmeyer

2

1 reference

Europe PubMed Central

29 March 2018

http://europepmc.org/abstract/MED/17092367

publication date

1 November 2006

1 reference

Europe PubMed Central

29 March 2018

http://europepmc.org/abstract/MED/17092367

British Journal of Nutrition

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Europe PubMed Central

29 March 2018

http://europepmc.org/abstract/MED/17092367

96

1 reference

Europe PubMed Central

29 March 2018

http://europepmc.org/abstract/MED/17092367

5

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Europe PubMed Central

29 March 2018

http://europepmc.org/abstract/MED/17092367

803-810

1 reference

Europe PubMed Central

29 March 2018

http://europepmc.org/abstract/MED/17092367

Molecular mechanism for silencing virally transduced genes involves histone deacetylation and chromatin condensation

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Butyrate is only one of several growth inhibitors produced during gut flora-mediated fermentation of dietary fibre sources

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Rationale for the luminal provision of butyrate in intestinal diseases

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Genes modulated by histone acetylation as new effectors of butyrate activity

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Butyrate may enhance toxicological defence in primary, adenoma and tumor human colon cells by favourably modulating expression of glutathione S-transferases genes, an approach in nutrigenomics

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The gut fermentation product butyrate, a chemopreventive agent, suppresses glutathione S-transferase theta (hGSTT1) and cell growth more in human colon adenoma (LT97) than tumor (HT29) cells

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Butyrate specifically modulates MUC gene expression in intestinal epithelial goblet cells deprived of glucose.

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Expression of glutathione S-transferases (GSTs) in human colon cells and inducibility of GSTM2 by butyrate

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Butyrate induces glutathione S-transferase in human colon cells and protects from genetic damage by 4-hydroxy-2-nonenal

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Dietary fibre and colorectal adenoma in a colorectal cancer early detection programme

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Butyrate upregulates stromelysin-1 production by intestinal mesenchymal cells.

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The short-chain fatty acid butyrate induces apoptosis in colorectal tumour cell lines.

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Transient vs. prolonged histone hyperacetylation: effects on colon cancer cell growth, differentiation, and apoptosis

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based on heuristic

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The effects of short-chain fatty acids on human colon cancer cell phenotype are associated with histone hyperacetylation.

1 reference

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Histone deacetylase inhibition selectively alters the activity and expression of cell cycle proteins leading to specific chromatin acetylation and antiproliferative effects

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7 January 2021

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Butyrate and markers of neoplastic change in the colon.

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Effect of transgalactooligosaccharides on the composition of the human intestinal microflora and on putative risk markers for colon cancer

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Short-chain fatty acids and human colonic function: roles of resistant starch and nonstarch polysaccharides

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Identifiers

10.1017/BJN20061948

1 reference

Europe PubMed Central

29 March 2018

http://europepmc.org/abstract/MED/17092367

1 reference

Europe PubMed Central

29 March 2018

http://europepmc.org/abstract/MED/17092367

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