(Q51295065)

2 April 2018

title

Oxidative DNA damage and activation of c-Jun N-terminal kinase pathway in fibroblasts from patients with hereditary spastic paraplegia. (English)

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2 April 2018

author

Franca Esposito

4

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2 April 2018

author name string

Andreina Milano

1

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2 April 2018

Nicola Montesano Gesualdi

2

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2 April 2018

Raffaele Teperino

3

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2 April 2018

Sergio Cocozza

5

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2 April 2018

Paola Ungaro

6

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2 April 2018

publication date

1 December 2005

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2 April 2018

Cellular and Molecular Neurobiology

published in

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2 April 2018

volume

25

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2 April 2018

issue

8

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2 April 2018

page(s)

1245-1254

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2 April 2018

https://scigraph.springernature.com/pub.10.1007/s10571-005-8501-2

exact match

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cites work

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Damage and repair of nerve cell DNA in toxic stress

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Up-regulation of c-Jun N-terminal kinase pathway in Friedreich's ataxia cells

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Loss of m-AAA protease in mitochondria causes complex I deficiency and increased sensitivity to oxidative stress in hereditary spastic paraplegia

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Oxidative stress and genetics in the pathogenesis of Parkinson's disease

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DNA damaging agents induce expression of Fas ligand and subsequent apoptosis in T lymphocytes via the activation of NF-kappa B and AP-1.

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Frataxin activates mitochondrial energy conversion and oxidative phosphorylation

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12 December 2020

Normal oxidative damage to mitochondrial and nuclear DNA is extensive

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12 December 2020

Increased DNA oxidation and decreased levels of repair products in Alzheimer's disease ventricular CSF.

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12 December 2020

MEKK1/JNK signaling stabilizes and activates p53.

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12 December 2020

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12 December 2020

The Friedreich's ataxia mutation confers cellular sensitivity to oxidant stress which is rescued by chelators of iron and calcium and inhibitors of apoptosis

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High-dose combination alkylating agents with autologous bone-marrow support in patients with breast cancer: preliminary assessment of DNA damage in individual peripheral blood lymphocytes using the single cell gel electrophoresis assay

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DNA base-excision repair enzyme apurinic/apyrimidinic endonuclease/redox factor-1 is increased and competent in the brain and spinal cord of individuals with amyotrophic lateral sclerosis.

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Differential activation of ERK and JNK mitogen-activated protein kinases by Raf-1 and MEKK

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Mitochondrial complex I inhibitor rotenone induces apoptosis through enhancing mitochondrial reactive oxygen species production

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12 December 2020

10.1007/S10571-005-8501-2

Identifiers

DOI

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2 April 2018

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2 April 2018