(Q51387360)

4 April 2018

title

Biphasic response after brain death induction: prominent part of catecholamines release in this phenomenon. (English)

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4 April 2018

9

Lehot JJ

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4 April 2018

author name string

Chiari P

1

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4 April 2018

Hadour G

2

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4 April 2018

Michel P

3

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4 April 2018

Piriou V

4

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4 April 2018

Rodriguez C

5

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4 April 2018

Budat C

6

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4 April 2018

Ovize M

7

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4 April 2018

Jegaden O

8

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4 April 2018

Ferrera R

10

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4 April 2018

publication date

1 July 2000

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4 April 2018

The Journal of Heart and Lung Transplantation

published in

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4 April 2018

volume

19

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4 April 2018

issue

7

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4 April 2018

page(s)

675-682

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4 April 2018

Hormonal and hemodynamic changes in a validated animal model of brain death.

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Cardiocirculatory effects of acutely increased intracranial pressure and subsequent brain death

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Desensitization of myocardial beta-adrenergic receptors and deterioration of left ventricular function after brain death

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Brain death further promotes ischemic reperfusion injury of the rabbit myocardium.

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Myocardial effects of experimental acute brain death: evaluation by hemodynamic and biological studies

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High performance liquid chromatography: a rapid isocratic method for determination of creatine compounds and adenine nucleotides in myocardial tissue

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Changes in hemodynamic and metabolic parameters following induced brain death in the pig

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Brain-death-induced cardiac contractile dysfunction: studies of possible neurohormonal and blood-borne mediators.

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Prevention of myocardial injury during brain death by total cardiac sympathectomy in the Chacma baboon.

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Variable effects of explosive or gradual increase of intracranial pressure on myocardial structure and function.

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Brain death-induced impairment of cardiac contractile performance can be reversed by explantation and may not preclude the use of hearts for transplantation

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Myocardial contractility in a canine model of the brain-dead organ donor

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Myocardial β-Adrenergic Receptor Function and High-Energy Phosphates in Brain Death– Related Cardiac Dysfunction

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The influence of thyroid hormone replacement in a porcine brain death model.

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Cardiovascular consequences of severe hypophosphataemia in brain-dead patients

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Circulating cardiac troponin T in potential heart transplant donors

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Ultrastructure changes associated with brain death in the human donor heart.

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7 January 2021

Triiodothyronine reverses depressed contractile performance after excessive catecholamine stimulation.

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7 January 2021

Catecholamine cardiotoxicity

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Cardiac beta-adrenergic neuroeffector systems in acute myocardial dysfunction related to brain injury. Evidence for catecholamine-mediated myocardial damage

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Myocardial dysfunction in donor hearts. A possible etiology

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Catecholamines can induce adenosine receptor-mediated protection of the myocardium but do not participate in ischemic preconditioning in the rabbit

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7 January 2021

alpha 1-adrenergic agonists precondition rabbit ischemic myocardium independent of adenosine by direct activation of protein kinase C

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7 January 2021

Estimation of myocardial interstitial norepinephrine release after brain death using cardiac microdialysis.

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7 January 2021

Left Ventricular Wall Motion Abnormalities in Subarachnoid Hemorrhage: An Echocardiographic Study

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7 January 2021

Left ventricular wall motion abnormalities in patients with subarachnoid hemorrhage: neurogenic stunned myocardium

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7 January 2021

10.1016/S1053-2498(00)00127-3

Identifiers

DOI

1 reference

4 April 2018

1 reference

4 April 2018