(Q51579259)

7 April 2018

title

Insulin action on glucose transport and plasma membrane GLUT4 content in skeletal muscle from patients with NIDDM. (English)

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7 April 2018

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7 April 2018

Anna Gumà

object named as

A Gumà

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author name string

L He

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7 April 2018

E Odegoard Wahlström

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A Klip

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7 April 2018

H Wallberg-Henriksson

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publication date

1 October 1996

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7 April 2018

published in

Diabetologia

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7 April 2018

volume

39

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7 April 2018

page(s)

1180-1189

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issue

10

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https://scigraph.springernature.com/pub.10.1007/bf02658504

exact match

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cites work

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Increased glutamine:fructose-6-phosphate amidotransferase activity in skeletal muscle of patients with NIDDM.

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Pre-exposure to glucosamine induces insulin resistance of glucose transport and glycogen synthesis in isolated rat skeletal muscles. Study of mechanisms in muscle and in rat-1 fibroblasts overexpressing the human insulin receptor

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Hyperglycemia Activates Glucose Transport in Rat Skeletal Muscle Via a Ca2+-Dependent Mechanism

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Acute hyperglycemia provides an insulin-independent inducer for GLUT4 translocation in C2C12 myotubes and rat skeletal muscle

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Glucose-free fatty acid cycle operates in human heart and skeletal muscle in vivo

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Gene expression of GLUT4 in skeletal muscle from insulin-resistant patients with obesity, IGT, GDM, and NIDDM

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Hyperglycemia normalizes insulin-stimulated skeletal muscle glucose oxidation and storage in noninsulin-dependent diabetes mellitus

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Glucose transport activity in skeletal muscles from transgenic mice overexpressing GLUT1. Increased basal transport is associated with a defective response to diverse stimuli that activate GLUT4.

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Prevention by protein kinase C inhibitors of glucose-induced insulin-receptor tyrosine kinase resistance in rat fat cells

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Evidence against altered expression of GLUT1 or GLUT4 in skeletal muscle of patients with obesity or NIDDM.

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Indirect effect of catecholamines on development of insulin resistance in skeletal muscle from diabetic rats.

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Reversibility of decreased insulin-stimulated glucose transport capacity in diabetic muscle with in vitro incubation. Insulin is not required

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Differential regulation of intracellular glucose metabolism by glucose and insulin in human muscle

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Subcellular distribution of GLUT 4 in the skeletal muscle of lean type 2 (non-insulin-dependent) diabetic patients in the basal state

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Increased glucose effectiveness in normoglycemic but insulin-resistant relatives of patients with non-insulin-dependent diabetes mellitus. A novel compensatory mechanism

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Influence of physical training on formation of muscle capillaries in type I diabetes

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"Semi-open" muscle biopsy technique. A simple outpatient procedure

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Effect of Acute Hyperglycemia on Glucose Metabolism in Skeletal Muscles in IDDM Patients

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Untersuchungen uber die Harnstoffbildung im Tierkörper

1 reference

11 October 2022

https://opencitations.net/index/coci/api/v1/citations/10.1515/BCHM2.1932.210.1-2.33

Identifiers

DOI

10.1007/BF02658504

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7 April 2018

PubMed ID

8897005

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7 April 2018