(Q51670074)

English

Mnsod overexpression extends the yeast chronological (G(0)) life span but acts independently of Sir2p histone deacetylase to shorten the replicative life span of dividing cells.

scientific article published in June 2003

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scholarly article

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4 November 2019

Mnsod overexpression extends the yeast chronological (G(0)) life span but acts independently of Sir2p histone deacetylase to shorten the replicative life span of dividing cells (English)

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4 November 2019

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4 November 2019

Morag J Maclean

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4 November 2019

Pedro Moradas-Ferreira

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4 November 2019

Mehdi Mollapour

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object named as

Mehdi Mollapour

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4 November 2019

6

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Peter W Piper

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4 November 2019

author name string

Nicholas Harris

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4 November 2019

publication date

1 June 2003

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4 November 2019

Free Radical Biology and Medicine

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4 November 2019

34

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4 November 2019

1599-1606

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4 November 2019

12

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4 November 2019

FLP recombinase-mediated induction of Cu/Zn-superoxide dismutase transgene expression can extend the life span of adult Drosophila melanogaster flies

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A cytosolic catalase is needed to extend adult lifespan in C. elegans daf-C and clk-1 mutants.

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Evolution of ageing

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Adaptive response of the yeast Saccharomyces cerevisiae to reactive oxygen species: defences, damage and death

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Extrachromosomal rDNA circles--a cause of aging in yeast

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Calendar life span versus budding life span of Saccharomyces cerevisiae

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Regulation of longevity and stress resistance by Sch9 in yeast

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Base excision repair activities required for yeast to attain a full chronological life span

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Inhibition of cell growth in NIH/3T3 fibroblasts by overexpression of manganese superoxide dismutase: mechanistic studies

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PCR-synthesis of marker cassettes with long flanking homology regions for gene disruptions in S. cerevisiae

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Loss of prohibitins, though it shortens the replicative life span of yeast cells undergoing division, does not shorten the chronological life span of G0-arrested cells.

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Catalase in vitro

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10 Superoxide dismutase assays

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Regulation of manganese superoxide dismutase in Saccharomyces cerevisiae. The role of respiratory chain activity

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Stress signaling in yeast.

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The mitochondrial cytochrome c peroxidase Ccp1 of Saccharomyces cerevisiae is involved in conveying an oxidative stress signal to the transcription factor Pos9 (Skn7).

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Null mutants of Saccharomyces cerevisiae Cu,Zn superoxide dismutase: characterization and spontaneous mutation rates

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Chimeras of human extracellular and intracellular superoxide dismutases. Analysis of structure and function of the individual domains

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On the developmental theory of ageing. I. starvation resistance and longevity in Drosophila melanogaster in relation to pre-adult breeding conditions.

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Effect of Cu,Zn superoxide dismutase disruption mutation on replicative senescence in Saccharomyces cerevisiae.

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The influence of oxygen toxicity on yeast mother cell-specific aging

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Elimination of replication block protein Fob1 extends the life span of yeast mother cells.

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Mechanisms of cellular senescence

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Calorie restriction extends Saccharomyces cerevisiae lifespan by increasing respiration

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High osmolarity extends life span in Saccharomyces cerevisiae by a mechanism related to calorie restriction

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Requirement of NAD and SIR2 for life-span extension by calorie restriction in Saccharomyces cerevisiae

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The shortened replicative life span of prohibitin mutants of yeast appears to be due to defective mitochondrial segregation in old mother cells

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Divide and multiply: organelle partitioning in yeast

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A superoxide dismutase mimic protects sodA sodB Escherichia coli against aerobic heating and stationary-phase death.

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Superoxide dismutase activity is essential for stationary phase survival in Saccharomyces cerevisiae. Mitochondrial production of toxic oxygen species in vivo

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Identifiers

10.1016/S0891-5849(03)00210-7

1 reference

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4 November 2019

1 reference

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4 November 2019

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