(Q51916582)

English

Long-term facilitation of nociceptive withdrawal reflexes following low-frequency conditioning electrical stimulation: a new model for central sensitization in humans.

scientific article published on 27 January 2010

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scholarly article

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Europe PubMed Central

17 April 2018

http://europepmc.org/abstract/MED/20110183

Long-term facilitation of nociceptive withdrawal reflexes following low-frequency conditioning electrical stimulation: a new model for central sensitization in humans. (English)

1 reference

Europe PubMed Central

17 April 2018

http://europepmc.org/abstract/MED/20110183

central sensitization

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based on heuristic

inferred from title

Carsten Dahl Mørch

2

1 reference

Europe PubMed Central

17 April 2018

http://europepmc.org/abstract/MED/20110183

Ole Kæseler Andersen

3

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Europe PubMed Central

17 April 2018

http://europepmc.org/abstract/MED/20110183

José A Biurrun Manresa

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José A Biurrun Manresa

1

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publication date

27 January 2010

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Europe PubMed Central

17 April 2018

http://europepmc.org/abstract/MED/20110183

European Journal of Pain

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Europe PubMed Central

17 April 2018

http://europepmc.org/abstract/MED/20110183

14

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Europe PubMed Central

17 April 2018

http://europepmc.org/abstract/MED/20110183

8

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Europe PubMed Central

17 April 2018

http://europepmc.org/abstract/MED/20110183

822-831

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Europe PubMed Central

17 April 2018

http://europepmc.org/abstract/MED/20110183

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1 reference

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21 January 2018

Evidence for central summation of C and A delta nociceptive activity in man.

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Gradual enlargement of human withdrawal reflex receptive fields following repetitive painful stimulation.

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Differential effect of supraspinal modulation on the nociceptive withdrawal reflex and pain sensation.

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Catastrophizing is related to pain ratings, but not nociceptive flexion reflex threshold

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Capsaicin-induced central facilitation of a nociceptive flexion reflex in humans

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Preferential stimulation of Adelta fibers by intra-epidermal needle electrode in humans

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Central sensitization and LTP: do pain and memory share similar mechanisms?

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Mechanisms of inflammatory pain

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Perceptual correlates of nociceptive long-term potentiation and long-term depression in humans.

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Human surrogate models of neuropathic pain

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Effects of the NMDA-receptor antagonist ketamine on perceptual correlates of long-term potentiation within the nociceptive system.

1 reference

https://api.crossref.org/works/10.1016%2FJ.EJPAIN.2009.12.008

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The role of heterosynaptic facilitation in long-term potentiation (LTP) of human pain sensation

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A new model of electrically evoked pain and hyperalgesia in human skin: the effects of intravenous alfentanil, S(+)-ketamine, and lidocaine

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Teager-Kaiser energy operation of surface EMG improves muscle activity onset detection.

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A new interpretation of nonlinear energy operator and its efficacy in spike detection.

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Mechanisms underlying induction of homosynaptic long-term depression in area CA1 of the hippocampus

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Evidence suggesting a transcortical pathway from cutaneous foot afferents to tibialis anterior motoneurones in man.

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Differential inhibitory effect on human nociceptive skin senses induced by local stimulation of thin cutaneous fibers

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Cellular memory in spinal nociceptive circuitry

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Learning and memory in pain pathways

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Understanding LTP in pain pathways

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Induction of long-term potentiation at spinal synapses by noxious stimulation or nerve injury.

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The lower limb flexion reflex in humans

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Which nerve fibers mediate the axon reflex flare in human skin?

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Long term potentiation of single WDR neurons in spinalized rats.

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Mental stress inhibits pain perception and heart rate variability but not a nociceptive withdrawal reflex

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Peripheral and central mechanisms of cutaneous hyperalgesia

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Supraspinal influences on nociceptive flexion reflex and pain sensation in man

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Identifiers

10.1016/J.EJPAIN.2009.12.008

1 reference

Europe PubMed Central

17 April 2018

http://europepmc.org/abstract/MED/20110183

1 reference

Europe PubMed Central

17 April 2018

http://europepmc.org/abstract/MED/20110183

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