(Q52025498)

20 April 2018

title

Spatial learning in transgenic mice expressing human presenilin 1 (PS1) transgenes. (English)

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20 April 2018

author name string

Janus C

1

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20 April 2018

D'Amelio S

2

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20 April 2018

Amitay O

3

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20 April 2018

Chishti MA

4

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20 April 2018

Strome R

5

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20 April 2018

Fraser P

6

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20 April 2018

Carlson GA

7

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20 April 2018

Roder JC

8

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20 April 2018

St George-Hyslop P

9

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20 April 2018

Westaway D

10

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20 April 2018

publication date

1 July 2000

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20 April 2018

published in

Neurobiology of Aging

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20 April 2018

volume

21

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20 April 2018

issue

4

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20 April 2018

page(s)

541-549

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20 April 2018

Cognitive and neurobiologic markers of early Alzheimer disease

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Transgenic mice with Alzheimer presenilin 1 mutations show accelerated neurodegeneration without amyloid plaque formation.

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Mutant presenilins of Alzheimer's disease increase production of 42-residue amyloid beta-protein in both transfected cells and transgenic mice

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Increased amyloid-beta42(43) in brains of mice expressing mutant presenilin 1

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Stress-induced disturbances in Morris water-maze performance: interstrain variability.

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Fibril formation by primate, rodent, and Dutch-hemorrhagic analogues of Alzheimer amyloid beta-protein

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Transgenic mouse brain histopathology resembles early Alzheimer's disease

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Place navigation impaired in rats with hippocampal lesions

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Formation of beta-amyloid protein deposits in brains of transgenic mice

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Transgenic mice expressing hamster prion protein produce species-specific scrapie infectivity and amyloid plaques.

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Biochemistry of altered brain proteins in Alzheimer's disease

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Alzheimer's disease: genotypes, phenotypes, and treatments

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7 January 2021

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Identifiers

DOI

1 reference

20 April 2018

1 reference

20 April 2018