(Q52528383)

2 May 2018

title

Atm selectively regulates distinct p53-dependent cell-cycle checkpoint and apoptotic pathways. (English)

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2 May 2018

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Barlow C

1

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2 May 2018

Brown KD

2

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2 May 2018

Deng CX

3

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2 May 2018

Tagle DA

4

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2 May 2018

Wynshaw-Boris A

5

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2 May 2018

language of work or name

English

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publication date

1 December 1997

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2 May 2018

published in

Nature Genetics

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2 May 2018

volume

17

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2 May 2018

page(s)

453-456

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2 May 2018

issue

4

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2 May 2018

https://scigraph.springernature.com/pub.10.1038/ng1297-453

exact match

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cites work

A mutant p53 that discriminates between p53-responsive genes cannot induce apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

A mammalian cell cycle checkpoint pathway utilizing p53 and GADD45 is defective in ataxia-telangiectasia

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

Differential induction of transcriptionally active p53 following UV or ionizing radiation: defects in chromosome instability syndromes?

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

Dual roles of ATM in the cellular response to radiation and in cell growth control.

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

Ataxia telangiectasia mutant protein activates c-Abl tyrosine kinase in response to ionizing radiation

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

p53: puzzle and paradigm

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

p53, the cellular gatekeeper for growth and division

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

Tumor spectrum analysis in p53-mutant mice

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

Mice lacking p21CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint control

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

Atm-deficient mice: a paradigm of ataxia telangiectasia

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

Testing the role of p53 in the expression of genetic instability and apoptosis in ataxia-telangiectasia

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

atm and p53 cooperate in apoptosis and suppression of tumorigenesis, but not in resistance to acute radiation toxicity

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

Radiation-induced cell cycle arrest compromised by p21 deficiency

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

p53 is required for radiation-induced apoptosis in mouse thymocytes

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

Thymocyte apoptosis induced by p53-dependent and independent pathways.

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

Acid sphingomyelinase-deficient human lymphoblasts and mice are defective in radiation-induced apoptosis.

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

The product of the ATM gene is a 370-kDa nuclear phosphoprotein

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

The ataxia-telangiectasia gene product, a constitutively expressed nuclear protein that is not up-regulated following genome damage

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

WAF1, a potential mediator of p53 tumor suppression

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

A novel form of Epstein-Barr virus latency in normal B cells in vivo

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

p53 levels, functional domains, and DNA damage determine the extent of the apoptotic response of tumor cells

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

Sequence-specific transcriptional activation is essential for growth suppression by p53.

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

Improved method for computing potential doubling time from flow cytometric data

1 reference

https://api.crossref.org/works/10.1038%2FNG1297-453

7 January 2021

Identifiers

DOI

10.1038/NG1297-453

1 reference

2 May 2018

Dimensions Publication ID

0 references

1 reference

2 May 2018