(Q52534683)

2 May 2018

title

Antagonism of cytotoxic T-lymphocyte activation by soluble CD8. (English)

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2 May 2018

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1

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author name string

Gerth UC

2

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Price DA

3

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Purbhoo MA

4

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Boulter JM

5

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Gao GF

6

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Bell JI

7

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Phillips RE

8

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Jakobsen BK

9

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language of work or name

English

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publication date

1 April 1999

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2 May 2018

published in

Nature Medicine

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volume

5

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issue

4

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page(s)

399-404

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2 May 2018

https://scigraph.springernature.com/pub.10.1038/7398

exact match

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cites work

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Role of the major histocompatibility complex class I antigens in tumor growth and metastasis

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Effector mechanisms in allograft rejection

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CD8 is needed for development of cytotoxic T cells but not helper T cells

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Requirement for CD8-major histocompatibility complex class I interaction in positive and negative selection of developing T cells

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The CD8 coreceptor revisited: one chain good, two chains better

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Interaction of the unique N-terminal region of tyrosine kinase p56lck with cytoplasmic domains of CD4 and CD8 is mediated by cysteine motifs

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T cell receptor and CD8-dependent tyrosine phosphorylation events in cytotoxic T lymphocytes: activation of p56lck by CD8 binding to class I protein

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Fidelity of T cell activation through multistep T cell receptor zeta phosphorylation

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Both high and low avidity antibodies to the T cell receptor can have agonist or antagonist activity

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Requirement for association of p56lck with CD4 in antigen-specific signal transduction in T cells

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Cytotoxic T-lymphocyte activation involves a cascade of signalling and adhesion events

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Activated CD8 binding to class I protein mediated by the T-cell receptor results in signalling

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CD8 enhances formation of stable T-cell receptor/MHC class I molecule complexes

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Antagonism of cytotoxic T lymphocyte-mediated lysis by natural HIV-1 altered peptide ligands requires simultaneous presentation of agonist and antagonist peptides.

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Natural variants of cytotoxic epitopes are T-cell receptor antagonists for antiviral cytotoxic T cells

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Altered hapten ligands antagonize trinitrophenyl-specific cytotoxic T cells and block internalization of hapten-specific receptors

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A TCR binds to antagonist ligands with lower affinities and faster dissociation rates than to agonists

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CD4 augments the response of a T cell to agonist but not to antagonist ligands

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Initiation of signal transduction through the T cell receptor requires the multivalent engagement of peptide/MHC ligands [corrected]

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7 January 2021

Disruption of CD8-dependent negative and positive selection of thymocytes is correlated with a decreased association between CD8 and the protein tyrosine kinase, p56lck

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7 January 2021

Phosphorylation of murine CD8α is not essential for responses of T cell hybridomas to antigen

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T cell receptor and coreceptor CD8 alphaalpha bind peptide-MHC independently and with distinct kinetics

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7 January 2021

CD8 independence and specificity of cytotoxic T lymphocytes restricted by HLA-Aw68. 1

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7 January 2021

A structure-based approach to designing synthetic CD8alpha peptides that can inhibit cytotoxic T-lymphocyte responses

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7 January 2021

Soluble interleukin-2 receptor and soluble CD8 antigen in active rheumatoid arthritis

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7 January 2021

Identifiers

DOI

10.1038/7398

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2 May 2018

Dimensions Publication ID

1031549960

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2 May 2018