(Q52576782)

3 May 2018

title

Endogenous RGS proteins modulate SA and AV nodal functions in isolated heart: implications for sick sinus syndrome and AV block. (English)

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3 May 2018

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5

Richard R Neubig

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3 May 2018

author name string

Ying Fu

1

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3 May 2018

Xinyan Huang

2

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3 May 2018

Lin Piao

3

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3 May 2018

Anatoli N Lopatin

4

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3 May 2018

publication date

2 February 2007

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3 May 2018

American Journal of Physiology Heart and Circulatory Physiology

published in

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3 May 2018

volume

292

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3 May 2018

issue

5

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3 May 2018

page(s)

H2532-9

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3 May 2018

N-terminal residues control proteasomal degradation of RGS2, RGS4, and RGS5 in human embryonic kidney 293 cells

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RGS proteins reconstitute the rapid gating kinetics of gbetagamma-activated inwardly rectifying K+ channels

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The bee venom peptide tertiapin underlines the role of I(KACh) in acetylcholine-induced atrioventricular blocks

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Altered cardiovascular responses in mice lacking the M(1) muscarinic acetylcholine receptor

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G-protein signaling participates in the development of diabetic cardiomyopathy

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Homozygous SCN5A mutation in long-QT syndrome with functional two-to-one atrioventricular block

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Dominant-negative suppression of IK1 in the mouse heart leads to altered cardiac excitability

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Cardiac-specific overexpression of RhoA results in sinus and atrioventricular nodal dysfunction and contractile failure

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RGS8 accelerates G-protein-mediated modulation of K+ currents

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Regulator of G-protein signaling-2 mediates vascular smooth muscle relaxation and blood pressure

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10.1152/AJPHEART.01391.2006

21 January 2018

Identifiers

DOI

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3 May 2018

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3 May 2018