(Q52659036)

5 May 2018

title

MicroRNA-29a induces apoptosis via increasing the Bax:Bcl-2 ratio in dermal fibroblasts of patients with systemic sclerosis. (English)

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5 May 2018

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3

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5 May 2018

author name string

Saeideh Jafarinejad-Farsangi

1

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5 May 2018

Ali Farazmand

2

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5 May 2018

Farhad Gharibdoost

4

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5 May 2018

Elham Karimizadeh

5

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5 May 2018

Farshid Noorbakhsh

6

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5 May 2018

Habibeh Faridani

7

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5 May 2018

Ahmad Reza Jamshidi

8

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5 May 2018

publication date

10 April 2015

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5 May 2018

published in

Autoimmunity

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5 May 2018

volume

48

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5 May 2018

issue

6

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5 May 2018

page(s)

369-378

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5 May 2018

Mechanisms of skin fibrosis in systemic sclerosis

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Systemic sclerosis: a prototypic multisystem fibrotic disorder

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Fibroblasts in fibrosis: novel roles and mediators

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Apoptosis and myofibroblasts in the pathogenesis of systemic sclerosis

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Apoptosis and cancer: the genesis of a research field

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Apoptosis: a basic biological phenomenon with wide-ranging implications in human disease

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Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics

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Clearance of dying cells and autoimmunity

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Bcl-2-family proteins and the role of mitochondria in apoptosis

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Inhibition of mechanosensitive signaling in myofibroblasts ameliorates experimental pulmonary fibrosis

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The relationship between BcI2, Bax and p53: consequences for cell cycle progression and cell death.

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Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

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How do BCL-2 proteins induce mitochondrial outer membrane permeabilization?

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Bcl-2 is overexpressed and alters the threshold for apoptosis in a cholangiocarcinoma cell line

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Bax forms multispanning monomers that oligomerize to permeabilize membranes during apoptosis

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Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c.

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Most mammalian mRNAs are conserved targets of microRNAs

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The role of microRNAs in skin fibrosis.

21 January 2018

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The emerging role of circulating microRNAs as biomarkers in autoimmune diseases

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MicroRNA-29, a key regulator of collagen expression in systemic sclerosis

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TGF-β/Smad3 signaling promotes renal fibrosis by inhibiting miR-29

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miR-29 inhibits bleomycin-induced pulmonary fibrosis in mice.

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miR-29b as a therapeutic agent for angiotensin II-induced cardiac fibrosis by targeting TGF-β/Smad3 signaling

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Effects of microRNA-29 on apoptosis, tumorigenicity, and prognosis of hepatocellular carcinoma

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MicroRNA-21 in scleroderma fibrosis and its function in TGF-β-regulated fibrosis-related genes expression

21 January 2018

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The extracellular matrix: an active or passive player in fibrosis?

21 January 2018

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Micro-RNA profiling reveals a role for miR-29 in human and murine liver fibrosis

21 January 2018

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Dysregulation of microRNAs after myocardial infarction reveals a role of miR-29 in cardiac fibrosis

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Suppression of type I collagen production by microRNA-29b in cultured human stellate cells

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Renal medullary microRNAs in Dahl salt-sensitive rats: miR-29b regulates several collagens and related genes

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Suppression of microRNA-29 expression by TGF-β1 promotes collagen expression and renal fibrosis

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MicroRNA-29 family, a crucial therapeutic target for fibrosis diseases

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Resistance to caspase-independent cell death requires persistence of intact mitochondria

21 January 2018

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https://api.crossref.org/works/10.3109%2F08916934.2015.1030616

The rheostat in the membrane: BCL-2 family proteins and apoptosis

21 January 2018

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https://api.crossref.org/works/10.3109%2F08916934.2015.1030616

Bcl-XL is qualitatively different from and ten times more effective than Bcl-2 when expressed in a breast cancer cell line

21 January 2018

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Novel small-molecule inhibitors of Bcl-XL to treat lung cancer.

21 January 2018

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https://api.crossref.org/works/10.3109%2F08916934.2015.1030616

Transformation-dependent susceptibility of rat hepatic stellate cells to apoptosis induced by soluble Fas ligand

21 January 2018

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Hepatocyte-specific disruption of Bcl-xL leads to continuous hepatocyte apoptosis and liver fibrotic responses.

21 January 2018

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The deletion of transforming growth factor-beta-induced myofibroblasts depends on growth conditions and actin organization

21 January 2018

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https://api.crossref.org/works/10.3109%2F08916934.2015.1030616

Tumor necrosis factor alpha inhibits type I collagen synthesis through repressive CCAAT/enhancer-binding proteins

21 January 2018

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https://api.crossref.org/works/10.3109%2F08916934.2015.1030616

NF-kappaB inhibits expression of the alpha1(I) collagen gene.

21 January 2018

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https://api.crossref.org/works/10.3109%2F08916934.2015.1030616

TNF-α increases cardiac fibroblast lysyl oxidase expression through TGF-β and PI3Kinase signaling pathways.

21 January 2018

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https://api.crossref.org/works/10.3109%2F08916934.2015.1030616

Myocardial extracellular matrix remodeling in transgenic mice overexpressing tumor necrosis factor alpha can be modulated by anti-tumor necrosis factor alpha therapy

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F08916934.2015.1030616

Diverse biological functions of extracellular collagen processing enzymes

21 January 2018

1 reference

https://api.crossref.org/works/10.3109%2F08916934.2015.1030616

Apoptosis and molecular targeting therapy in cancer

21 January 2018

1 reference

12 January 2024

https://opencitations.net/index/api/v1/citations/10.1155/2014/150845

Identifiers

DOI

10.3109/08916934.2015.1030616

1 reference

5 May 2018

1 reference

5 May 2018