(Q53209168)

12 May 2018

title

Two distinct ubiquitin immunoreactive senile plaques in Alzheimer's disease: relationship with the intellectual status in 29 cases. (English)

1 reference

12 May 2018

1 reference

6

J J Hauw

1 reference

12 May 2018

Charles Duyckaerts

3

object named as

C Duyckaerts

1 reference

12 May 2018

author name string

Y He

1

1 reference

12 May 2018

P Delaère

2

1 reference

12 May 2018

M Wasowicz

4

1 reference

12 May 2018

F Piette

5

1 reference

12 May 2018

publication date

1 January 1993

1 reference

12 May 2018

published in

Acta Neuropathologica

1 reference

12 May 2018

volume

86

1 reference

12 May 2018

issue

1

1 reference

12 May 2018

page(s)

109-116

1 reference

12 May 2018

https://scigraph.springernature.com/pub.10.1007/bf00454909

exact match

0 references

cites work

About the presence of paired helical filaments in dystrophic neurites participating in the plaque formation.

1 reference

12 December 2020

The association between quantitative measures of dementia and of senile change in the cerebral grey matter of elderly subjects.

1 reference

12 December 2020

Ubiquitination and abnormal phosphorylation of paired helical filaments in Alzheimer's disease

1 reference

12 December 2020

Neurofibrillary tangles of Alzheimer's disease: an immunohistochemical study

1 reference

12 December 2020

The "normal" brain. "Abnormal" ubiquitinilated deposits highlight an age-related protein change

1 reference

12 December 2020

Ubiquitin immunoreactive structures in normal human brains. Distribution and developmental aspects

1 reference

12 December 2020

Laminar distribution of neocortical senile plaques in senile dementia of the Alzheimer type.

1 reference

12 December 2020

Subtypes and differential laminar distributions of ?A4 deposits in Alzheimer's disease: relationship with the intellectual status of 26 cases

1 reference

12 December 2020

Rating of the lesions in senile dementia of the Alzheimer type: concordance between laboratories. A European multicenter study under the auspices of EURAGE.

1 reference

12 December 2020

Ubiquitin immunoelectron microscopy of dystrophic neurites in cerebellar senile plaques of Alzheimer's disease

1 reference

12 December 2020

Ubiquitin is a common factor in intermediate filament inclusion bodies of diverse type in man, including those of Parkinson's disease, Pick's disease, and Alzheimer's disease, as well as Rosenthal fibres in cerebellar astrocytomas, cytoplasmic bodie

1 reference

12 December 2020

Heterogeneity of ubiquitin immunoreactivity in neurofibrillary tangles of Alzheimer's disease.

1 reference

12 December 2020

Neurofibrillary Tangles, Dystrophic IMeurites (Curly Fibers), and Abnormal Phosphorylation of Tau

1 reference

12 December 2020

Microtubule-associated polypeptides tau are altered in Alzheimer paired helical filaments

1 reference

12 December 2020

Alzheimer's disease: Tau proteins, the promoting factors of microtubule assembly, are major components of paired helical filaments.

1 reference

12 December 2020

Ubiquitin is detected in neurofibrillary tangles and senile plaque neurites of Alzheimer disease brains

1 reference

12 December 2020

Ubiquitinated protein conjugates are specifically enriched in the lysosomal system of fibroblasts

1 reference

12 December 2020

Abnormal phosphorylation of tau precedes ubiquitination in neurofibrillary pathology of Alzheimer disease

1 reference

12 December 2020

Ubiquitin is a component of neurofibrillary tangles in a variety of neurodegenerative diseases

1 reference

12 December 2020

Fibre disorganization in the neocortex of patients with senile dementia of the Alzheimer type

1 reference

12 December 2020

A68: a major subunit of paired helical filaments and derivatized forms of normal Tau.

1 reference

12 December 2020

Lack of ubiquitin immunoreactivities at both ends of neuropil threads. Possible bidirectional growth of neuropil threads

1 reference

12 December 2020

Ubiquitin is associated with abnormal cytoplasmic filaments characteristic of neurodegenerative diseases

1 reference

12 December 2020

Quantitative assessment of the density of neurofibrillary tangles and senile plaques in senile dementia of the Alzheimer type. Comparison of immunocytochemistry with a specific antibody and Bodian's protargol method.

1 reference

12 December 2020

Ubiquitin has intrinsic proteolytic activity: implications for cellular regulation

1 reference

12 December 2020

Cortical atrophy in senile dementia of the Alzheimer type is mainly due to a decrease in cortical length

1 reference

12 December 2020

Does amyloid precede paired helical filaments in the senile plaque? A study of 15 cases with graded intellectual status in aging and Alzheimer disease

1 reference

12 December 2020

Deposition of ?/A4 protein along neuronal plasma membranes in diffuse senile plaques

1 reference

12 December 2020

Ultrastructure of the neuropil threads in the Alzheimer brain: their dendritic origin and accumulation in the senile plaques

1 reference

12 December 2020

Accumulation of abnormally phosphorylated tau precedes the formation of neurofibrillary tangles in Alzheimer's disease

1 reference

12 December 2020

Alzheimer patients and Down patients: abnormal presynaptic terminals are related to cerebral preamyloid deposits.

1 reference

12 December 2020

Microtubule-associated protein tau. A component of Alzheimer paired helical filaments.

1 reference

12 December 2020

A68 proteins in Alzheimer's disease are composed of several tau isoforms in a phosphorylated state which affects their electrophoretic mobilities

1 reference

12 December 2020

Identification of normal and pathological aging in prospectively studied nondemented elderly humans

1 reference

12 December 2020

Large amounts of neocortical beta A4 deposits without neuritic plaques nor tangles in a psychometrically assessed, non-demented person

1 reference

12 December 2020

Neuropil threads of Alzheimer's disease show a marked alteration of the normal cytoskeleton

1 reference

12 December 2020

Ubiquitin is a component of paired helical filaments in Alzheimer's disease.

1 reference

12 December 2020

Tau, paired helical filaments and amyloid in the neocortex: a morphometric study of 15 cases with graded intellectual status in aging and senile dementia of Alzheimer type

1 reference

12 December 2020

Identifiers

DOI

10.1007/BF00454909

1 reference

12 May 2018

PubMed ID

8396836

1 reference

12 May 2018