(Q53473485)

14 May 2018

title

Interaction with XIAP prevents full caspase-3/-7 activation in proliferating human T lymphocytes. (English)

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14 May 2018

author name string

Maren Paulsen

1

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14 May 2018

Sandra Ussat

2

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14 May 2018

Marten Jakob

3

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14 May 2018

Gudrun Scherer

4

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14 May 2018

Inga Lepenies

5

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14 May 2018

Stefan Schütze

6

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14 May 2018

Dieter Kabelitz

7

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14 May 2018

Sabine Adam-Klages

8

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14 May 2018

language of work or name

English

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publication date

1 July 2008

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14 May 2018

European Journal of Immunology

published in

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14 May 2018

volume

38

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14 May 2018

issue

7

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14 May 2018

page(s)

1979-1987

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14 May 2018

Caspase-8 serves both apoptotic and nonapoptotic roles

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1 reference

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Effector CD4+ T cells generate intermediate caspase activity and cleavage of caspase-8 substrates

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Caspase inhibition blocks human T cell proliferation by suppressing appropriate regulation of IL-2, CD25, and cell cycle-associated proteins

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Apoptosis-independent functions of killer caspases.

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Death receptors and caspases: role in lymphocyte proliferation, cell death, and autoimmunity

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7 January 2021

Evidence for CPP32 activation in the absence of apoptosis during T lymphocyte stimulation

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7 January 2021

Activation of caspase-3-like enzymes in non-apoptotic T cells

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7 January 2021

Early activation of caspases during T lymphocyte stimulation results in selective substrate cleavage in nonapoptotic cells

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7 January 2021

Caspase activation is required for T cell proliferation

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Pleiotropic defects in lymphocyte activation caused by caspase-8 mutations lead to human immunodeficiency

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Essential role for caspase 8 in T-cell homeostasis and T-cell-mediated immunity

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7 January 2021

Different subcellular distribution of caspase-3 and caspase-7 following Fas-induced apoptosis in mouse liver

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7 January 2021

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7 January 2021

Redistribution of activated caspase-3 to the nucleus during butyric acid-induced apoptosis

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7 January 2021

Spatio-temporal activation of caspase revealed by indicator that is insensitive to environmental effects

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7 January 2021

Nuclear translocation of caspase-3 is dependent on its proteolytic activation and recognition of a substrate-like protein(s).

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7 January 2021

A-kinase-anchoring protein 95 functions as a potential carrier for the nuclear translocation of active caspase 3 through an enzyme-substrate-like association

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7 January 2021

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7 January 2021

Granzyme release and caspase activation in activated human T-lymphocytes.

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7 January 2021

Death receptors leave a caspase footprint that Smacs of XIAP.

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7 January 2021

Bcl-2 and Bcl-xL inhibit CD95-mediated apoptosis by preventing mitochondrial release of Smac/DIABLO and subsequent inactivation of X-linked inhibitor-of-apoptosis protein

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7 January 2021

Pro-caspase-3 is a major physiologic target of caspase-8.

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7 January 2021

Discovery of embelin as a cell-permeable, small-molecular weight inhibitor of XIAP through structure-based computational screening of a traditional herbal medicine three-dimensional structure database

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7 January 2021

Characterization of XIAP-deficient mice

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7 January 2021

Generation and characterization of Smac/DIABLO-deficient mice.

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7 January 2021

XIAP deficiency in humans causes an X-linked lymphoproliferative syndrome

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7 January 2021

Essential contribution of caspase 3/CPP32 to apoptosis and its associated nuclear changes

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https://api.crossref.org/works/10.1002%2FEJI.200838211

7 January 2021

Caspases 3 and 7: key mediators of mitochondrial events of apoptosis

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7 January 2021

Sensitization to death receptor cytotoxicity by inhibition of fas-associated death domain protein (FADD)/caspase signaling. Requirement of cell cycle progression

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7 January 2021

Identifiers

DOI

10.1002/EJI.200838211

1 reference

14 May 2018

1 reference

14 May 2018