(Q53521856)

15 May 2018

title

Effect of chemically modified IL-13 short interfering RNA on development of airway hyperresponsiveness in mice. (English)

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15 May 2018

10

Erwin W Gelfand

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15 May 2018

author name string

Tricia N Lively

1

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15 May 2018

Karl Kossen

2

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15 May 2018

Annette Balhorn

3

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Toshiyuki Koya

4

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Shawn Zinnen

5

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Katsuyuki Takeda

6

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15 May 2018

Joseph J Lucas

7

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15 May 2018

Barry Polisky

8

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15 May 2018

Ivan M Richards

9

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15 May 2018

language of work or name

English

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publication date

22 October 2007

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15 May 2018

The Journal of Allergy and Clinical Immunology

published in

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15 May 2018

volume

121

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issue

1

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15 May 2018

page(s)

88-94

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15 May 2018

Effector CD8+ T cells mediate inflammation and airway hyper-responsiveness

cites work

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Requirement for IL-13 independently of IL-4 in experimental asthma

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Mast cells, Fc epsilon RI, and IL-13 are required for development of airway hyperresponsiveness after aerosolized allergen exposure in the absence of adjuvant

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Potent and persistent in vivo anti-HBV activity of chemically modified siRNAs

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Mitogen-activated protein kinase activation through Fc epsilon receptor I and stem cell factor receptor is differentially regulated by phosphatidylinositol 3-kinase and calcineurin in mouse bone marrow-derived mast cells

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Inhibition of influenza virus production in virus-infected mice by RNA interference

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Interleukin-13 in asthma pathogenesis.

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Direct effects of interleukin-13 on signaling pathways for physiological responses in cultured human airway smooth muscle cells.

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Establishment of mouse cell lines which constitutively secrete large quantities of interleukin 2, 3, 4 or 5, using modified cDNA expression vectors

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Critical role for IL-13 in the development of allergen-induced airway hyperreactivity

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Airway hyperresponsiveness: a story of mice and men and cytokines

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Inhibition of respiratory syncytial virus infection with intranasal siRNA nanoparticles targeting the viral NS1 gene

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Adoptive transfer of T cells induces airway hyperresponsiveness independently of airway eosinophilia but in a signal transducer and activator of transcription 6-dependent manner

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Interleukin-13: central mediator of allergic asthma

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Therapeutic silencing of an endogenous gene by systemic administration of modified siRNAs

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Activity of stabilized short interfering RNA in a mouse model of hepatitis B virus replication

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The importance of leukotrienes in airway inflammation in a mouse model of asthma

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Using siRNA in prophylactic and therapeutic regimens against SARS coronavirus in Rhesus macaque

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Comparison between cationic polymers and lipids in mediating systemic gene delivery to the lungs

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Small interfering RNA targeting heme oxygenase-1 enhances ischemia-reperfusion-induced lung apoptosis.

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Interleukin-13 mediates airways hyperreactivity through the IL-4 receptor-alpha chain and STAT-6 independently of IL-5 and eotaxin

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Sequence-specific potent induction of IFN-alpha by short interfering RNA in plasmacytoid dendritic cells through TLR7

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12 December 2020

Development of eosinophilic airway inflammation and airway hyperresponsiveness in mast cell-deficient mice

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Protein kinase C alpha, betaI, and betaII isozymes regulate cytokine production in mast cells through MEKK2/ERK5-dependent and -independent pathways

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Pulmonary expression of interleukin-13 causes inflammation, mucus hypersecretion, subepithelial fibrosis, physiologic abnormalities, and eotaxin production

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Suppression of discoidin domain receptor 1 by RNA interference attenuates lung inflammation

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Position-specific chemical modification of siRNAs reduces "off-target" transcript silencing

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Synthesis, deprotection, analysis and purification of RNA and ribozymes

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Interleukin 4, but not interleukin 5 or eosinophils, is required in a murine model of acute airway hyperreactivity.

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The role of IL-13 in established allergic airway disease.

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10.1016/J.JACI.2007.08.029

Identifiers

DOI

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15 May 2018

PubMed ID

17936889

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15 May 2018