(Q53907457)

English

The redox state of endogenous pyridine nucleotides can determine both the degree of mitochondrial oxidative stress and the solute selectivity of the permeability transition pore.

scientific article

In more languages

Statements

scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

22 May 2018

http://europepmc.org/abstract/MED/10922464

The redox state of endogenous pyridine nucleotides can determine both the degree of mitochondrial oxidative stress and the solute selectivity of the permeability transition pore. (English)

1 reference

Europe PubMed Central

PubMed publication ID

22 May 2018

http://europepmc.org/abstract/MED/10922464

author name string

Zago EB

1

1 reference

Europe PubMed Central

PubMed publication ID

22 May 2018

http://europepmc.org/abstract/MED/10922464

Castilho RF

2

1 reference

Europe PubMed Central

PubMed publication ID

22 May 2018

http://europepmc.org/abstract/MED/10922464

Vercesi AE

3

1 reference

Europe PubMed Central

PubMed publication ID

22 May 2018

http://europepmc.org/abstract/MED/10922464

language of work or name

0 references

publication date

1 July 2000

1 reference

Europe PubMed Central

PubMed publication ID

22 May 2018

http://europepmc.org/abstract/MED/10922464

1 reference

Europe PubMed Central

PubMed publication ID

22 May 2018

http://europepmc.org/abstract/MED/10922464

478

1 reference

Europe PubMed Central

PubMed publication ID

22 May 2018

http://europepmc.org/abstract/MED/10922464

1-2

1 reference

Europe PubMed Central

PubMed publication ID

22 May 2018

http://europepmc.org/abstract/MED/10922464

29-33

1 reference

Europe PubMed Central

PubMed publication ID

22 May 2018

http://europepmc.org/abstract/MED/10922464

The participation of pyridine nucleotides redox state and reactive oxygen in the fatty acid-induced permeability transition in rat liver mitochondria.

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

The participation of reactive oxygen species and protein thiols in the mechanism of mitochondrial inner membrane permeabilization by calcium plus prooxidants.

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

Effect of inorganic phosphate concentration on the nature of inner mitochondrial membrane alterations mediated by Ca2+ ions. A proposed model for phosphate-stimulated lipid peroxidation.

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

Physiological roles of nicotinamide nucleotide transhydrogenase

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

Mitochondria are excitable organelles capable of generating and conveying electrical and calcium signals.

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

From calcium signaling to cell death: two conformations for the mitochondrial permeability transition pore. Switching from low- to high-conductance state

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

Modulation of inner mitochondrial membrane channel activity

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

The mitochondrial permeability transition.

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

Permeability transition pore of the inner mitochondrial membrane can operate in two open states with different selectivities

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

Safranine as a probe of the mitochondrial membrane potential

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

Interactions of cyclophilin with the mitochondrial inner membrane and regulation of the permeability transition pore, and cyclosporin A-sensitive channel.

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

On the involvement of a cyclosporin A sensitive mitochondrial pore in myocardial reperfusion injury

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

High protonic potential actuates a mechanism of production of reactive oxygen species in mitochondria

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

Role of uncoupled and non-coupled oxidations in maintenance of safely low levels of oxygen and its one-electron reductants

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

Mitochondrial calcium transport

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

Intracellular adenosine triphosphate (ATP) concentration: a switch in the decision between apoptosis and necrosis

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

21 January 2018

Mitochondrial damage induced by conditions of oxidative stress

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

7 January 2021

Isolated cerebral and cerebellar mitochondria produce free radicals when exposed to elevated CA2+ and Na+: implications for neurodegeneration

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

7 January 2021

Permeabilization of the inner mitochondrial membrane by Ca2+ ions is stimulated by t-butyl hydroperoxide and mediated by reactive oxygen species generated by mitochondria

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

7 January 2021

The thiol-specific antioxidant enzyme prevents mitochondrial permeability transition. Evidence for the participation of reactive oxygen species in this mechanism

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

7 January 2021

Ca2+-induced increased lipid packing and domain formation in submitochondrial particles. A possible early step in the mechanism of Ca2+-stimulated generation of reactive oxygen species by the respiratory chain

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

7 January 2021

t-Butylhydroperoxide-induced Ca2+ efflux from liver mitochondria in the presence of physiological concentrations of Mg2+ and ATP

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

7 January 2021

The participation of NADP, the transmembrane potential and the energy-linked NAD(P) transhydrogenase in the process of Ca2+ efflux from rat liver mitochondria

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

7 January 2021

The mitochondrial permeability transition: from biochemical curiosity to pathophysiological mechanism

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

7 January 2021

Dissociation of NAD(P)+-stimulated mitochondrial Ca2+ efflux from swelling and membrane damage

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

7 January 2021

Fluorescence of nicotinamide adenine dinucleotides during the active transport of Ca 2+ ions in liver mitochondria

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

7 January 2021

Inhibition of mitochondrial nicotinamide nucleotide transhydrogenase by CoA-thioesters of long-chain fatty acids

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

7 January 2021

Relationships between the NAD(P) redox state, fatty acid oxidation, and inner membrane permeability in rat liver mitochondria

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

7 January 2021

Cyclosporin A, but not FK 506, protects mitochondria and neurons against hypoglycemic damage and implicates the mitochondrial permeability transition in cell death.

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2800%2901815-9

7 January 2021

Identifiers

10.1016/S0014-5793(00)01815-9

1 reference

Europe PubMed Central

PubMed publication ID

22 May 2018

http://europepmc.org/abstract/MED/10922464

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

22 May 2018

http://europepmc.org/abstract/MED/10922464

Sitelinks

Wikipedia(0 entries)

Wikibooks(0 entries)

Wikinews(0 entries)

Wikiquote(0 entries)

Wikisource(0 entries)

Wikiversity(0 entries)

Wikivoyage(0 entries)

Wiktionary(0 entries)

Multilingual sites(0 entries)

Retrieved from "https://www.wikidata.org/w/index.php?title=Q53907457&oldid=1373122490"