(Q53922251)

22 May 2018

title

Defective neural tube morphogenesis and altered apoptosis in the absence of both JNK1 and JNK2. (English)

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22 May 2018

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6

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22 May 2018

author name string

Sabapathy K

1

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22 May 2018

Jochum W

2

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22 May 2018

Hochedlinger K

3

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22 May 2018

Chang L

4

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22 May 2018

Karin M

5

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22 May 2018

publication date

1 December 1999

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22 May 2018

Mechanisms of Development

published in

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22 May 2018

volume

89

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22 May 2018

issue

1-2

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22 May 2018

page(s)

115-124

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22 May 2018

Amino-terminal phosphorylation of c-Jun regulates stress-induced apoptosis and cellular proliferation

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Defective T Cell Differentiation in the Absence of Jnk1

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Functions of c-Jun in liver and heart development

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splotch (Sp2H), a mutation affecting development of the mouse neural tube, shows a deletion within the paired homeodomain of Pax-3

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MEKKs, GCKs, MLKs, PAKs, TAKs, and tpls: upstream regulators of the c-Jun amino-terminal kinases?

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Embryonic death of Mek1-deficient mice reveals a role for this kinase in angiogenesis in the labyrinthine region of the placenta

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Selective interaction of JNK protein kinase isoforms with transcription factors

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Differential requirement for caspase 9 in apoptotic pathways in vivo.

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c-Jun is essential for normal mouse development and hepatogenesis

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Abnormal morphogenesis but intact IKK activation in mice lacking the IKKalpha subunit of IkappaB kinase

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Normal mouse strains differ in the site of initiation of closure of the cranial neural tube

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c-Jun can recruit JNK to phosphorylate dimerization partners via specific docking interactions

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JNK2 contains a specificity-determining region responsible for efficient c-Jun binding and phosphorylation

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The regulation of AP-1 activity by mitogen-activated protein kinases

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Transcriptional control by protein phosphorylation: signal transmission from the cell surface to the nucleus

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The Jnk1 and Jnk2 protein kinases are required for regional specific apoptosis during early brain development

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7 January 2021

Reduced apoptosis and cytochrome c-mediated caspase activation in mice lacking caspase 9

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Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice

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Withdrawal of survival factors results in activation of the JNK pathway in neuronal cells leading to Fas ligand induction and cell death

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7 January 2021

Severe liver degeneration in mice lacking the IkappaB kinase 2 gene

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7 January 2021

Identification of a dual specificity kinase that activates the Jun kinases and p38-Mpk2

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Specificity of receptor tyrosine kinase signaling: transient versus sustained extracellular signal-regulated kinase activation

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JNK2 is required for efficient T-cell activation and apoptosis but not for normal lymphocyte development.

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Neurulation in the mouse: manner and timing of neural tube closure

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7 January 2021

Role of SAPK/ERK kinase-1 in the stress-activated pathway regulating transcription factor c-Jun

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7 January 2021

Transcription factor AP-2 essential for cranial closure and craniofacial development

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7 January 2021

Mitogen-activated protein kinase kinase 7 is an activator of the c-Jun NH2-terminal kinase

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7 January 2021

Molecular cloning and characterization of human JNKK2, a novel Jun NH2-terminal kinase-specific kinase

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7 January 2021

Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene

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7 January 2021

Targeted disruption of the MKK4 gene causes embryonic death, inhibition of c-Jun NH2-terminal kinase activation, and defects in AP-1 transcriptional activity

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7 January 2021

Apaf1 is required for mitochondrial pathways of apoptosis and brain development

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7 January 2021

Prenatal folic acid treatment suppresses acrania and meroanencephaly in mice mutant for the Cart1 homeobox gene

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7 January 2021

10.1016/S0925-4773(99)00213-0

Identifiers

DOI

1 reference

22 May 2018

1 reference

22 May 2018