(Q54135016)

25 May 2018

title

Dietary restriction augments protection against induction of the mitochondrial permeability transition. (English)

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25 May 2018

author name string

B S Kristal

series ordinal

1

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25 May 2018

B P Yu

series ordinal

2

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25 May 2018

publication date

1 May 1998

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25 May 2018

Free Radical Biology and Medicine

published in

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25 May 2018

volume

24

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25 May 2018

issue

7-8

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25 May 2018

page(s)

1269-1277

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25 May 2018

Mitochondrial calcium transport: physiological and pathological relevance

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Recent progress on regulation of the mitochondrial permeability transition pore; a cyclosporin-sensitive pore in the inner mitochondrial membrane

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'Pore' formation is not required for the hydroperoxide-induced Ca2+ release from rat liver mitochondria

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Ca2+-dependent NAD(P)+-induced alterations of rat liver and hepatoma mitochondrial membrane permeability.

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Cyclosporin A-sensitive and insensitive mechanisms produce the permeability transition in mitochondria

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Permeabilization of the inner mitochondrial membrane by Ca2+ ions is stimulated by t-butyl hydroperoxide and mediated by reactive oxygen species generated by mitochondria

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Enhanced induction of the mitochondrial permeability transition following acute menadione administration

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Channel-specific induction of the cyclosporine A-sensitive mitochondrial permeability transition by menadione

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4-Hydroxyhexenal is a potent inducer of the mitochondrial permeability transition

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Oxidative damage of mitochondria induced by Fe(II)citrate or t-butyl hydroperoxide in the presence of Ca2+: effect of coenzyme Q redox state

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Recruitment of mitochondrial cyclophilin to the mitochondrial inner membrane under conditions of oxidative stress that enhance the opening of a calcium-sensitive non-specific channel

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Further evidence that cyclosporin A protects mitochondria from calcium overload by inhibiting a matrix peptidyl-prolyl cis-trans isomerase. Implications for the immunosuppressive and toxic effects of cyclosporin

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Evidence for the presence of a reversible Ca2+-dependent pore activated by oxidative stress in heart mitochondria

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Mechanisms by which mitochondria transport calcium

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Kinetic evidence for a heart mitochondrial pore activated by Ca2+, inorganic phosphate and oxidative stress. A potential mechanism for mitochondrial dysfunction during cellular Ca2+ overload

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Aging and oxidative stress: modulation by dietary restriction

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7 January 2021

Abnormalities in the mitochondrial permeability transition in diabetic rats.

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7 January 2021

Effect of spermine on mitochondrial glutathione release

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7 January 2021

Effect of chronic food restriction in aging rats. I. Liver subcellular membranes.

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7 January 2021

Effect of diet restriction on some biochemical parameters related to aging in mice.

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7 January 2021

Oxidative damage, mitochondrial oxidant generation and antioxidant defenses during aging and in response to food restriction in the mouse

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7 January 2021

Anti-lipoperoxidation action of food restriction

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7 January 2021

Detoxification of reactive aldehydes in mitochondria: effects of age and dietary restriction

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7 January 2021

Dietary modulation of DNA damage in human

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7 January 2021

Cellular defenses against damage from reactive oxygen species

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7 January 2021

Effect of chronic food restriction in aging rats. II. Liver cytosolic antioxidants and related enzymes.

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7 January 2021

Influences of dietary restriction and age on liver enzyme activities and lipid peroxidation in mice.

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7 January 2021

Characterization of age-related malondialdehyde oxidation: the effect of modulation by food restriction

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7 January 2021

10.1016/S0891-5849(97)00444-9

Identifiers

DOI

1 reference

25 May 2018

1 reference

25 May 2018