(Q54566777)

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N-cadherin mediates angiogenesis by regulating monocyte chemoattractant protein-1 expression via PI3K/Akt signaling in prostate cancer cells.

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Europe PubMed Central

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1 June 2018

http://europepmc.org/abstract/MED/21855541

N-cadherin mediates angiogenesis by regulating monocyte chemoattractant protein-1 expression via PI3K/Akt signaling in prostate cancer cells. (English)

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Europe PubMed Central

PubMed publication ID

1 June 2018

http://europepmc.org/abstract/MED/21855541

prostate cancer

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based on heuristic

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Jasti S Rao

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Europe PubMed Central

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1 June 2018

http://europepmc.org/abstract/MED/21855541

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Arun Kumar Nalla

1

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Europe PubMed Central

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1 June 2018

http://europepmc.org/abstract/MED/21855541

Norman Estes

2

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Europe PubMed Central

PubMed publication ID

1 June 2018

http://europepmc.org/abstract/MED/21855541

Jitendra Patel

3

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1 June 2018

http://europepmc.org/abstract/MED/21855541

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publication date

9 August 2011

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Europe PubMed Central

PubMed publication ID

1 June 2018

http://europepmc.org/abstract/MED/21855541

Experimental Cell Research

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Europe PubMed Central

PubMed publication ID

1 June 2018

http://europepmc.org/abstract/MED/21855541

317

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Europe PubMed Central

PubMed publication ID

1 June 2018

http://europepmc.org/abstract/MED/21855541

17

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Europe PubMed Central

PubMed publication ID

1 June 2018

http://europepmc.org/abstract/MED/21855541

2512-2521

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Europe PubMed Central

PubMed publication ID

1 June 2018

http://europepmc.org/abstract/MED/21855541

The role of epithelial-mesenchymal transition in cancer pathology

1 reference

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Twist overexpression correlates with hepatocellular carcinoma metastasis through induction of epithelial-mesenchymal transition.

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Expression of N-cadherin adhesion molecules associated with early morphogenetic events in chick development

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Purified N-cadherin is a potent substrate for the rapid induction of neurite outgrowth

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Differential localization of VE- and N-cadherins in human endothelial cells: VE-cadherin competes with N-cadherin for junctional localization

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Changes in cadherin-catenin complexes in the progression of human bladder carcinoma

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Disparate E-cadherin mutations in LCIS and associated invasive breast carcinomas

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Downregulation of N-cadherin expression inhibits invasiveness, arrests cell cycle and induces cell apoptosis in esophageal squamous cell carcinoma

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N-cadherin-mediated adhesion and aberrant catenin expression in anaplastic thyroid-carcinoma cell lines.

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Exogenous expression of N-cadherin in breast cancer cells induces cell migration, invasion, and metastasis

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Soluble N-cadherin fragment promotes angiogenesis

1 reference

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A switch from E-cadherin to N-cadherin expression indicates epithelial to mesenchymal transition and is of strong and independent importance for the progress of prostate cancer

1 reference

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Prostate cancer progression into androgen independency is associated with alterations in cell adhesion and invasivity

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N-Cadherin expression in human prostate carcinoma cell lines. An epithelial-mesenchymal transformation mediating adhesion withStromal cells.

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7 January 2021

Complex cadherin expression in human prostate cancer cells

1 reference

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The role of tumor microenvironment in prostate cancer bone metastasis

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Activation of MCP-1/CCR2 axis promotes prostate cancer growth in bone

1 reference

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7 January 2021

Akt-regulated pathways in prostate cancer

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N-cadherin switching occurs in high Gleason grade prostate cancer

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Metastatic patterns of prostate cancer: an autopsy study of 1,589 patients

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Role of cadherins in the transendothelial migration of melanoma cells in culture

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Cell-cell interactions during transendothelial migration of tumor cells

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The role of endothelial cells in tumor invasion and metastasis

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7 January 2021

N-cadherin acts upstream of VE-cadherin in controlling vascular morphogenesis

1 reference

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7 January 2021

Monocyte chemoattractant protein-1-induced angiogenesis is mediated by vascular endothelial growth factor-A

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Monocyte chemotactic protein (MCP)-1 promotes angiogenesis via a novel transcription factor, MCP-1-induced protein (MCPIP)

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Epithelial-mesenchymal transition in development and cancer: role of phosphatidylinositol 3' kinase/AKT pathways

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Novel expression of N-cadherin elicits in vitro bladder cell invasion via the Akt signaling pathway.

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7 January 2021

Phospho-Akt pathway activation and inhibition depends on N-cadherin or phospho-EGFR expression in invasive human bladder cancer cell lines.

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7 January 2021

AKT plays a central role in tumorigenesis

1 reference

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Phosphatidylinositol 3-kinase function is required for transforming growth factor beta-mediated epithelial to mesenchymal transition and cell migration.

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CD151 amplifies signaling by integrin α6β1 to PI3K and induces the epithelial-mesenchymal transition in HCC cells

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Twist regulates cytokine gene expression through a negative feedback loop that represses NF-kappaB activity

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HAb18G/CD147 promotes epithelial-mesenchymal transition through TGF-β signaling and is transcriptionally regulated by Slug

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Identifiers

10.1016/J.YEXCR.2011.07.024

1 reference

Europe PubMed Central

PubMed publication ID

1 June 2018

http://europepmc.org/abstract/MED/21855541

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

1 June 2018

http://europepmc.org/abstract/MED/21855541

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