(Q55043263)

16 June 2018

title

Granzyme K directly processes bid to release cytochrome c and endonuclease G leading to mitochondria-dependent cell death. (English)

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16 June 2018

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Tongbiao Zhao

1

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16 June 2018

Honglian Zhang

series ordinal

2

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16 June 2018

Yuming Guo

series ordinal

3

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16 June 2018

Zusen Fan

series ordinal

4

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16 June 2018

language of work or name

English

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publication date

16 February 2007

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16 June 2018

Journal of Biological Chemistry

published in

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16 June 2018

volume

282

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16 June 2018

issue

16

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16 June 2018

page(s)

12104-12111

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16 June 2018

The orphan granzymes of humans and mice

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Granzyme A and B-deficient killer lymphocytes are defective in eliciting DNA fragmentation but retain potent in vivo anti-tumor capacity

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Differential expression of human granzymes A, B, and K in natural killer cells and during CD8+ T cell differentiation in peripheral blood

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A natural killer cell granule protein that induces DNA fragmentation and apoptosis

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21 January 2018

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A mitochondrial perspective on cell death

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BAX and BAK regulation of endoplasmic reticulum Ca2+: a control point for apoptosis

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Granzyme A induces caspase-independent mitochondrial damage, a required first step for apoptosis

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Nuclear war: the granzyme A-bomb

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Granzyme B short-circuits the need for caspase 8 activity during granule-mediated cytotoxic T-lymphocyte killing by directly cleaving Bid.

21 January 2018

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Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

21 January 2018

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Tumor suppressor NM23-H1 is a granzyme A-activated DNase during CTL-mediated apoptosis, and the nucleosome assembly protein SET is its inhibitor

21 January 2018

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HMG2 interacts with the nucleosome assembly protein SET and is a target of the cytotoxic T-lymphocyte protease granzyme A

21 January 2018

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Cleaving the oxidative repair protein Ape1 enhances cell death mediated by granzyme A

21 January 2018

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Granzyme M directly cleaves inhibitor of caspase-activated DNase (CAD) to unleash CAD leading to DNA fragmentation

21 January 2018

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Caspase-mediated loss of mitochondrial function and generation of reactive oxygen species during apoptosis

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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

21 January 2018

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21 January 2018

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21 January 2018

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

21 January 2018

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Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

21 January 2018

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Endonuclease G is an apoptotic DNase when released from mitochondria

21 January 2018

1 reference

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Granzyme B induces BID-mediated cytochrome c release and mitochondrial permeability transition

21 January 2018

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Granzyme B-mediated cytochrome c release is regulated by the Bcl-2 family members bid and Bax.

21 January 2018

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Purification of three cytotoxic lymphocyte granule serine proteases that induce apoptosis through distinct substrate and target cell interactions

21 January 2018

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21 January 2018

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21 January 2018

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Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity

21 January 2018

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Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

21 January 2018

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Regulated targeting of BAX to mitochondria.

21 January 2018

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Cell damage-induced conformational changes of the pro-apoptotic protein Bak in vivo precede the onset of apoptosis

21 January 2018

1 reference

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A role for proapoptotic BID in the DNA-damage response

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M611006200

21 January 2018

Identifiers

DOI

10.1074/JBC.M611006200

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16 June 2018

PubMed ID

17308307

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16 June 2018