(Q55475202)

English

Detection of PTEN nonsense mutation and psiPTEN expression in central nervous system high-grade astrocytic tumors by a yeast-based stop codon assay.

scientific article published in September 2000

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scholarly article

1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

Detection of PTEN nonsense mutation and psiPTEN expression in central nervous system high-grade astrocytic tumors by a yeast-based stop codon assay. (English)

1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

author name string

C L Zhang

1

1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

M Tada

2

1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

H Kobayashi

3

1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

M Nozaki

4

1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

T Moriuchi

5

1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

H Abe

6

1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

language of work or name

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publication date

1 September 2000

1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

19

1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

4346-4353

1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

38

1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

https://scigraph.springernature.com/pub.10.1038/sj.onc.1203795

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PTEN/MMAC1 mutations in primary glioblastomas and short-term cultures of malignant gliomas.

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A highly conserved processed PTEN pseudogene is located on chromosome band 9p21

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Pten is essential for embryonic development and tumour suppression

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A simple p53 functional assay for screening cell lines, blood, and tumors

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Growth suppression of glioma cells by PTEN requires a functional phosphatase catalytic domain

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Protein kinase B (PKB/Akt) activity is elevated in glioblastoma cells due to mutation of the tumor suppressor PTEN/MMAC.

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Absence of p53 gene mutations in a tumor panel representative of pilocytic astrocytoma diversity using a p53 functional assay.

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Alterations of PTEN/MMAC1, a candidate tumor suppressor gene, and its homologue, PTH2, in small cell lung cancer cell lines

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Structural and functional evidence for the presence of tumor suppressor genes on the short arm of chromosome 10 in human gliomas.

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PTEN1 is frequently mutated in primary endometrial carcinomas

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PTEN, a putative protein tyrosine phosphatase gene mutated in human brain, breast, and prostate cancer

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Pseudogenes as a paradigm of neutral evolution

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Germline mutations of the PTEN gene in Cowden disease, an inherited breast and thyroid cancer syndrome

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The tumor suppressor, PTEN/MMAC1, dephosphorylates the lipid second messenger, phosphatidylinositol 3,4,5-trisphosphate

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Mutation spectrum and genotype-phenotype analyses in Cowden disease and Bannayan-Zonana syndrome, two hamartoma syndromes with germline PTEN mutation

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Germline mutations in PTEN are present in Bannayan-Zonana syndrome.

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DMBT1, a new member of the SRCR superfamily, on chromosome 10q25.3-26.1 is deleted in malignant brain tumours

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The lipid phosphatase activity of PTEN is critical for its tumor supressor function

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Identification of a human homolog of the Drosophila neuralized gene within the 10q25.1 malignant astrocytoma deletion region

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Negative regulation of PKB/Akt-dependent cell survival by the tumor suppressor PTEN

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Identification of a candidate tumour suppressor gene, MMAC1, at chromosome 10q23.3 that is mutated in multiple advanced cancers

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Homozygous p53 gene mutation in a radiation-induced glioblastoma 10 years after treatment for an intracranial germ cell tumor: case report.

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Inhibition of cell migration, spreading, and focal adhesions by tumor suppressor PTEN

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PTEN (MMAC1) mutations are frequent in primary glioblastomas (de novo) but not in secondary glioblastomas.

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The genetic basis of Cowden's syndrome: three novel mutations in PTEN/MMAC1/TEP1.

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Association of epidermal growth factor receptor gene amplification with loss of chromosome 10 in human glioblastoma multiforme

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Deletion mapping of chromosome 19 in human gliomas.

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Multiple methods of visualizing the yeast vacuole permit evaluation of its morphology and inheritance during the cell cycle

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High cancer susceptibility and embryonic lethality associated with mutation of the PTEN tumor suppressor gene in mice

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PTEN germ-line mutations in juvenile polyposis coli

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7 January 2021

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10.1038/SJ.ONC.1203795

1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

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1 reference

Europe PubMed Central

12 July 2018

http://europepmc.org/abstract/MED/10980610

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