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Cancer drugs to treat birth defects
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scholarly article
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title
Cancer drugs to treat birth defects
(English)
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author
Andrew Oliver Mungo Wilkie
object named as
Andrew O M Wilkie
series ordinal
1
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language of work or name
English
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publication date
September 2007
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published in
Nature Genetics
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volume
39
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issue
9
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page(s)
1057-1059
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1057-9
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exact match
https://scigraph.springernature.com/pub.10.1038/ng0907-1057
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cites work
RNA interference and inhibition of MEK-ERK signaling prevent abnormal skeletal phenotypes in a mouse model of craniosynostosis
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https://api.crossref.org/works/10.1038%2FNG0907-1057
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7 January 2021
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Apert syndrome results from localized mutations of FGFR2 and is allelic with Crouzon syndrome
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Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNG0907-1057
retrieved
7 January 2021
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Biochemical analysis of pathogenic ligand-dependent FGFR2 mutations suggests distinct pathophysiological mechanisms for craniofacial and limb abnormalities
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNG0907-1057
retrieved
7 January 2021
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Frequent activating FGFR2 mutations in endometrial carcinomas parallel germline mutations associated with craniosynostosis and skeletal dysplasia syndromes
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Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNG0907-1057
retrieved
7 January 2021
based on heuristic
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A Ser250Trp substitution in mouse fibroblast growth factor receptor 2 (Fgfr2) results in craniosynostosis
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNG0907-1057
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Abnormalities in cartilage and bone development in the Apert syndrome FGFR2(+/S252W) mouse.
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Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNG0907-1057
retrieved
7 January 2021
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inferred from DOI database lookup
Strategies for silencing human disease using RNA interference
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Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNG0907-1057
retrieved
7 January 2021
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Mechanisms underlying differential responses to FGF signaling
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reference URL
https://api.crossref.org/works/10.1038%2FNG0907-1057
retrieved
7 January 2021
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Bad bones, absent smell, selfish testes: the pleiotropic consequences of human FGF receptor mutations
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Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNG0907-1057
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Attenuation of signaling pathways stimulated by pathologically activated FGF-receptor 2 mutants prevents craniosynostosis.
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNG0907-1057
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
A model for the pharmacological treatment of crouzon syndrome
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Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNG0907-1057
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Structures of human MAP kinase kinase 1 (MEK1) and MEK2 describe novel noncompetitive kinase inhibition.
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Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNG0907-1057
retrieved
7 January 2021
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Targeting the ERK signaling pathway in cancer therapy
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Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNG0907-1057
retrieved
7 January 2021
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Hyperactive Ras in developmental disorders and cancer
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reference URL
https://api.crossref.org/works/10.1038%2FNG0907-1057
retrieved
7 January 2021
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BRAF mutation predicts sensitivity to MEK inhibition
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Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNG0907-1057
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7 January 2021
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Identifiers
DOI
10.1038/NG0907-1057
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PubMed ID
17728773
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