(Q56368315)

1

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F Nakajima

2

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S Shimizu

3

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A Ogasawara

4

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A Wanaka

5

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H Moriya

6

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T A Einhorn

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M Yamazaki

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T.A Einhorn

Growth factor regulation of fracture repair.

7

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language of work or name

English

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publication date

November 2001

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published in

Bone

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volume

29

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issue

5

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page(s)

458-66

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458-466

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cites work

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A recurrent mutation in the tyrosine kinase domain of fibroblast growth factor receptor 3 causes hypochondroplasia

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Regulation of fracture repair by growth factors

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Production of a standard closed fracture in laboratory animal bone

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Immunolocalization and expression of bone morphogenetic proteins 2 and 4 in fracture healing

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Effects of basic fibroblast growth factor on bone formation in vitro

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Fibroblast growth factor (FGF)-2 directly stimulates mature osteoclast function through activation of FGF receptor 1 and p42/p44 MAP kinase

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Cultured bovine bone cells synthesize basic fibroblast growth factor and store it in their extracellular matrix.

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Structural characterization and biological functions of fibroblast growth factor

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Enhanced expression of type I receptors for bone morphogenetic proteins during bone formation

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Jackson-Weiss and Crouzon syndromes are allelic with mutations in fibroblast growth factor receptor 2

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Predominant use of a particular alpha-chain in suppressor T cell hybridomas specific for keyhole limpet hemocyanin

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Differential expression of fibroblast growth factor receptor-1, -2, and -3 and syndecan-1, -2, and -4 in neonatal rat mandibular condyle and calvaria during osteogenic differentiation in vitro

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A common mutation in the fibroblast growth factor receptor 1 gene in Pfeiffer syndrome

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Recombinant human basic fibroblast growth factor accelerates fracture healing by enhancing callus remodeling in experimental dog tibial fracture

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Transient and localized expression of bone morphogenetic protein 4 messenger RNA during fracture healing

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Distinct and overlapping patterns of localization of bone morphogenetic protein (BMP) family members and a BMP type II receptor during fracture healing in rats

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Novel formulation of fibroblast growth factor-2 in a hyaluronan gel accelerates fracture healing in nonhuman primates

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Mutations in the transmembrane domain of FGFR3 cause the most common genetic form of dwarfism, achondroplasia

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FGF-mediated aspects of skeletal muscle growth and differentiation are controlled by a high affinity receptor, FGFR1

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Spatial and temporal expression of fibril-forming minor collagen genes (types V and XI) during fracture healing

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