Home
Random
Nearby
Log in
Settings
Donate
About Wikidata
Disclaimers
Search
(Q57082822)
Watch
English
Caspase activation is not death
article by Jean-Luc Perfettini & Guido Kroemer published April 2003 in Nature Immunology
In more languages
edit
Statements
instance of
scholarly article
0 references
title
Caspase activation is not death
(English)
0 references
author
Jean-Luc Perfettini
object named as
Jean-Luc Perfettini
series ordinal
1
0 references
Guido Kroemer
object named as
Guido Kroemer
series ordinal
2
0 references
language of work or name
English
0 references
publication date
April 2003
0 references
published in
Nature Immunology
0 references
volume
4
0 references
issue
4
0 references
page(s)
308-310
0 references
cites work
Caspase inhibition causes hyperacute tumor necrosis factor-induced shock via oxidative stress and phospholipase A2.
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNI0403-308
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
The mitochondrial death/life regulator in apoptosis and necrosis
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNI0403-308
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Inhibition of caspases increases the sensitivity of L929 cells to necrosis mediated by tumor necrosis factor.
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNI0403-308
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Sensitization to death receptor cytotoxicity by inhibition of fas-associated death domain protein (FADD)/caspase signaling. Requirement of cell cycle progression
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNI0403-308
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Structure/Function analysis of p55 tumor necrosis factor receptor and fas-associated death domain. Effect on necrosis in L929sA cells.
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNI0403-308
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNI0403-308
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Platelet formation is the consequence of caspase activation within megakaryocytes
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNI0403-308
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Caspase 3 activity is required for skeletal muscle differentiation
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNI0403-308
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Specific involvement of caspases in the differentiation of monocytes into macrophages
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNI0403-308
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Caspase activation is required for T cell proliferation
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FNI0403-308
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Identifiers
DOI
10.1038/NI0403-308
0 references
PubMed ID
12660728
0 references
Sitelinks
Wikipedia
(0 entries)
edit
Wikibooks
(0 entries)
edit
Wikinews
(0 entries)
edit
Wikiquote
(0 entries)
edit
Wikisource
(0 entries)
edit
Wikiversity
(0 entries)
edit
Wikivoyage
(0 entries)
edit
Wiktionary
(0 entries)
edit
Multilingual sites
(0 entries)
edit