(Q57243813)

1

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Inga Kadish

2

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Katja Wiesehan

3

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Susanne A. Funke

Phases of A beta-deposition in the human brain and its relevance for the development of AD.

4

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publication date

13 February 2009

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published in

ChemMedChem

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volume

4

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issue

2

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page(s)

276-282

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cites work

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Alzheimer Disease in the US Population

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Neuropathological stageing of Alzheimer-related changes

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Evolution of neuronal changes in the course of Alzheimer's disease.

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Alzheimer's disease: genes, proteins, and therapy

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Normal and abnormal tau neurobiology

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The morphological phenotype of beta-amyloid plaques and associated neuritic changes in Alzheimer's disease

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Amyloidogenic processing of beta-amyloid precursor protein in intracellular compartments

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Frequency of stages of Alzheimer-related lesions in different age categories

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Amyloid imaging in Alzheimer's disease

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Imaging brain amyloid in Alzheimer's disease with Pittsburgh Compound-B

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Transgenic mouse models of Alzheimer's disease: how useful have they been for therapeutic development?

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A decade of modeling Alzheimer's disease in transgenic mice.

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Selection of D-amino-acid peptides that bind to Alzheimer's disease amyloid peptide abeta1-42 by mirror image phage display

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Mirror-image phage display: aiming at the mirror

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Inhibition of cytotoxicity and amyloid fibril formation by a D-amino acid peptide that specifically binds to Alzheimer's disease amyloid peptide

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Deposition of mouse amyloid beta in human APP/PS1 double and single AD model transgenic mice.

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Comparison of the aggregation properties, secondary structure and apoptotic effects of wild-type, Flemish and Dutch N-terminally truncated amyloid beta peptides

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In vitro studies of Flemish, Dutch, and wild-type beta-amyloid provide evidence for two-staged neurotoxicity.

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Dutch, Flemish, Italian, and Arctic mutations of APP and resistance of Abeta to physiologically relevant proteolytic degradation.

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Diffuse amyloid deposition, but not plaque number, is reduced in amyloid precursor protein/presenilin 1 double-transgenic mice by pathway lesions.

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Evidence that synaptically released beta-amyloid accumulates as extracellular deposits in the hippocampus of transgenic mice.

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Axonal transport, amyloid precursor protein, kinesin-1, and the processing apparatus: revisited

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Disruption of corticocortical connections ameliorates amyloid burden in terminal fields in a transgenic model of Abeta amyloidosis.

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Consistent immunohistochemical detection of intracellular beta-amyloid42 in pyramidal neurons of Alzheimer's disease entorhinal cortex

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Intracellular accumulation of beta-amyloid(1-42) in neurons is facilitated by the alpha 7 nicotinic acetylcholine receptor in Alzheimer's disease.

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A peptide zipcode sufficient for anterograde transport within amyloid precursor protein

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3D-Reconstruction of microglia and amyloid in APP23 transgenic mice: no evidence of intracellular amyloid

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Transgenic AD model mice, effects of potential anti-AD treatments on inflammation and pathology

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Abeta42-driven cerebral amyloidosis in transgenic mice reveals early and robust pathology

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D-peptides as immunogens and diagnostic reagents

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Co-expression of multiple transgenes in mouse CNS: a comparison of strategies.

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Pathogenic effects of cerebral amyloid angiopathy mutations in the amyloid beta-protein precursor

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Transgenic mice expressing the human presenilin 1 gene demonstrate enhanced hippocampal reorganization following entorhinal cortex lesions

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Analysis of heterogeneous A4 peptides in human cerebrospinal fluid and blood by a newly developed sensitive Western blot assay.

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